6akp - Revision history

OCA at 09:36, 22 November 2023

OCA — Wed, 22 Nov 2023 09:36:14 GMT

←Older revision		Revision as of 09:36, 22 November 2023
Line 3:		Line 3:
	<StructureSection load='6akp' size='340' side='right'caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>		<StructureSection load='6akp' size='340' side='right'caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>
	== Structural highlights ==		== Structural highlights ==
-	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure with sequence from [https://en.wikipedia.org/wiki/ ~~] and [https://en.wikipedia.org/wiki/Human Human~~]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [https://proteopedia.org/fgij/fg.htm?mol=6AKP FirstGlance]. <br>	+	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure with sequence from [https://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [https://proteopedia.org/fgij/fg.htm?mol=6AKP FirstGlance]. <br>
-	</td></tr><tr id='~~ligand~~'><td class="sblockLbl"><b>[[~~Ligand~~\|~~Ligands~~:]]</b></td><td class="sblockDat" id="~~ligandDat~~">~~<scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene>~~</td></tr>	+	</td></tr><tr id='method'><td class="sblockLbl"><b>[[Empirical_models\|Method:]]</b></td><td class="sblockDat" id="methodDat">X-ray diffraction, [[Resolution\|Resolution]] 2.323Å</td></tr>
-	<tr id='~~gene~~'><td class="sblockLbl"><b>[[~~Gene~~\|~~Gene~~:]]</b></td><td class="sblockDat">~~FOXC2, FKHL14, MFH1 ([https://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?mode~~=~~Info&srchmode~~=~~5&id=9606 HUMAN])~~</td></tr>	+	<tr id='ligand'><td class="sblockLbl"><b>[[Ligand\|Ligands:]]</b></td><td class="sblockDat" id="ligandDat"><scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene></td></tr>
	<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [https://pdbe.org/6akp PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [https://www.ebi.ac.uk/pdbsum/6akp PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>		<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [https://pdbe.org/6akp PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [https://www.ebi.ac.uk/pdbsum/6akp PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>
	</table>		</table>
	== Disease ==		== Disease ==
-	[[https://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Defects in FOXC2 are the cause of lymphedema hereditary type 2 (LMPH2) [MIM:[https://omim.org/entry/153200 153200]]; also known as Meige lymphedema. Hereditary lymphedema is a chronic disabling condition which results in swelling of the extremities due to altered lymphatic flow. Patients with lymphedema suffer from recurrent local infections, and physical impairment.<ref>PMID:11078474</ref> Defects in FOXC2 are a cause of lymphedema-yellow nails (LYYN) [MIM:[https://omim.org/entry/153300 153300]]. LYYN is characterized by yellow, dystrophic, thick and slowly growing nails, associated with lymphedema and respiratory involvement. Lymphedema occurs more often in the lower limbs. It can appear at birth or later in life. Onset generally follows the onset of ungual abnormalities. Defects in FOXC2 are a cause of lymphedema-distichiasis (LYD) [MIM:[https://omim.org/entry/153400 153400]]. LYD is characterized by primary limb lymphedema usually starting at puberty (but in some cases later or at birth) and associated with distichiasis (double rows of eyelashes, with extra eyelashes growing from the Meibomian gland orifices).<ref>PMID:11499682</ref>	+	[https://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN] Defects in FOXC2 are the cause of lymphedema hereditary type 2 (LMPH2) [MIM:[https://omim.org/entry/153200 153200]; also known as Meige lymphedema. Hereditary lymphedema is a chronic disabling condition which results in swelling of the extremities due to altered lymphatic flow. Patients with lymphedema suffer from recurrent local infections, and physical impairment.<ref>PMID:11078474</ref> Defects in FOXC2 are a cause of lymphedema-yellow nails (LYYN) [MIM:[https://omim.org/entry/153300 153300]. LYYN is characterized by yellow, dystrophic, thick and slowly growing nails, associated with lymphedema and respiratory involvement. Lymphedema occurs more often in the lower limbs. It can appear at birth or later in life. Onset generally follows the onset of ungual abnormalities. Defects in FOXC2 are a cause of lymphedema-distichiasis (LYD) [MIM:[https://omim.org/entry/153400 153400]. LYD is characterized by primary limb lymphedema usually starting at puberty (but in some cases later or at birth) and associated with distichiasis (double rows of eyelashes, with extra eyelashes growing from the Meibomian gland orifices).<ref>PMID:11499682</ref>
	== Function ==		== Function ==
-	[[https://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Transcriptional activator. Might be involved in the formation of special mesenchymal tissues.<ref>PMID:9169153</ref>	+	[https://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN] Transcriptional activator. Might be involved in the formation of special mesenchymal tissues.<ref>PMID:9169153</ref>
	<div style="background-color:#fffaf0;">		<div style="background-color:#fffaf0;">
	== Publication Abstract from PubMed ==		== Publication Abstract from PubMed ==
Line 28:		Line 28:
	__TOC__		__TOC__
	</StructureSection>		</StructureSection>
-	[[Category: ~~Human~~]]	+	[[Category: Homo sapiens]]
	[[Category: Large Structures]]		[[Category: Large Structures]]
-	[[Category: Chen, X]]	+	[[Category: Chen X]]
-	[[Category: Chen, Y]]	+	[[Category: Chen Y]]
-	[[Category: Dai, S]]	+	[[Category: Dai S]]
-	[[Category: Guo, M]]	+	[[Category: Guo M]]
-	[[Category: Jiang, L]]	+	[[Category: Jiang L]]
-	[[Category: Li, J]]	+	[[Category: Li J]]
-	[[Category: Liang, X]]	+	[[Category: Liang X]]
-	[[Category: Wei, H]]	+	[[Category: Wei H]]
-	~~[[Category: Crystal structural]]~~	+
-	~~[[Category: Dna binding domain]]~~	+
-	~~[[Category: Dna binding protein-dna complex]]~~	+
-	~~[[Category: Dna recognition]]~~	+
-	~~[[Category: Foxc]]~~	+
-	~~[[Category: Lymphoedema distichiasis syndrome~~]]	+

OCA at 10:14, 18 March 2021

OCA — Thu, 18 Mar 2021 10:14:52 GMT

←Older revision		Revision as of 10:14, 18 March 2021
Line 3:		Line 3:
	<StructureSection load='6akp' size='340' side='right'caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>		<StructureSection load='6akp' size='340' side='right'caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>
	== Structural highlights ==		== Structural highlights ==
-	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure with sequence from [~~http~~://en.wikipedia.org/wiki/ ] and [~~http~~://en.wikipedia.org/wiki/Human Human]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [~~http~~://~~oca~~.~~weizmann.ac.il/oca-docs~~/fgij/fg.htm?mol=6AKP FirstGlance]. <br>	+	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure with sequence from [https://en.wikipedia.org/wiki/ ] and [https://en.wikipedia.org/wiki/Human Human]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [https://proteopedia.org/fgij/fg.htm?mol=6AKP FirstGlance]. <br>
-	</td></tr><tr id='ligand'><td class="sblockLbl"><b>[[Ligand\|Ligands:]]</b></td><td class="sblockDat"><scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene></td></tr>	+	</td></tr><tr id='ligand'><td class="sblockLbl"><b>[[Ligand\|Ligands:]]</b></td><td class="sblockDat" id="ligandDat"><scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene></td></tr>
-	<tr id='gene'><td class="sblockLbl"><b>[[Gene\|Gene:]]</b></td><td class="sblockDat">FOXC2, FKHL14, MFH1 ([~~http~~://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?mode=Info&srchmode=5&id=9606 HUMAN])</td></tr>	+	<tr id='gene'><td class="sblockLbl"><b>[[Gene\|Gene:]]</b></td><td class="sblockDat">FOXC2, FKHL14, MFH1 ([https://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?mode=Info&srchmode=5&id=9606 HUMAN])</td></tr>
-	<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[~~http~~://~~oca~~.~~weizmann.ac.il/oca-docs~~/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [~~http~~://pdbe.org/6akp PDBe], [~~http~~://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [~~http~~://www.ebi.ac.uk/pdbsum/6akp PDBsum], [~~http~~://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>	+	<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [https://pdbe.org/6akp PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [https://www.ebi.ac.uk/pdbsum/6akp PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>
	</table>		</table>
	== Disease ==		== Disease ==
-	[[~~http~~://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Defects in FOXC2 are the cause of lymphedema hereditary type 2 (LMPH2) [MIM:[~~http~~://omim.org/entry/153200 153200]]; also known as Meige lymphedema. Hereditary lymphedema is a chronic disabling condition which results in swelling of the extremities due to altered lymphatic flow. Patients with lymphedema suffer from recurrent local infections, and physical impairment.<ref>PMID:11078474</ref> Defects in FOXC2 are a cause of lymphedema-yellow nails (LYYN) [MIM:[~~http~~://omim.org/entry/153300 153300]]. LYYN is characterized by yellow, dystrophic, thick and slowly growing nails, associated with lymphedema and respiratory involvement. Lymphedema occurs more often in the lower limbs. It can appear at birth or later in life. Onset generally follows the onset of ungual abnormalities. Defects in FOXC2 are a cause of lymphedema-distichiasis (LYD) [MIM:[~~http~~://omim.org/entry/153400 153400]]. LYD is characterized by primary limb lymphedema usually starting at puberty (but in some cases later or at birth) and associated with distichiasis (double rows of eyelashes, with extra eyelashes growing from the Meibomian gland orifices).<ref>PMID:11499682</ref>	+	[[https://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Defects in FOXC2 are the cause of lymphedema hereditary type 2 (LMPH2) [MIM:[https://omim.org/entry/153200 153200]]; also known as Meige lymphedema. Hereditary lymphedema is a chronic disabling condition which results in swelling of the extremities due to altered lymphatic flow. Patients with lymphedema suffer from recurrent local infections, and physical impairment.<ref>PMID:11078474</ref> Defects in FOXC2 are a cause of lymphedema-yellow nails (LYYN) [MIM:[https://omim.org/entry/153300 153300]]. LYYN is characterized by yellow, dystrophic, thick and slowly growing nails, associated with lymphedema and respiratory involvement. Lymphedema occurs more often in the lower limbs. It can appear at birth or later in life. Onset generally follows the onset of ungual abnormalities. Defects in FOXC2 are a cause of lymphedema-distichiasis (LYD) [MIM:[https://omim.org/entry/153400 153400]]. LYD is characterized by primary limb lymphedema usually starting at puberty (but in some cases later or at birth) and associated with distichiasis (double rows of eyelashes, with extra eyelashes growing from the Meibomian gland orifices).<ref>PMID:11499682</ref>
	== Function ==		== Function ==
-	[[~~http~~://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Transcriptional activator. Might be involved in the formation of special mesenchymal tissues.<ref>PMID:9169153</ref>	+	[[https://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Transcriptional activator. Might be involved in the formation of special mesenchymal tissues.<ref>PMID:9169153</ref>
	<div style="background-color:#fffaf0;">		<div style="background-color:#fffaf0;">
	== Publication Abstract from PubMed ==		== Publication Abstract from PubMed ==
Line 21:		Line 21:
	</div>		</div>
	<div class="pdbe-citations 6akp" style="background-color:#fffaf0;"></div>		<div class="pdbe-citations 6akp" style="background-color:#fffaf0;"></div>
		+
		+	==See Also==
		+	*[[FOX 3D structures\|FOX 3D structures]]
	== References ==		== References ==
	<references/>		<references/>

OCA at 09:04, 1 May 2019

OCA — Wed, 01 May 2019 09:04:52 GMT

←Older revision		Revision as of 09:04, 1 May 2019
Line 1:		Line 1:

	==Crystal Structural of FOXC2 DNA binding domain bound to PC promoter==		==Crystal Structural of FOXC2 DNA binding domain bound to PC promoter==
-	<StructureSection load='6akp' size='340' side='right' caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>	+	<StructureSection load='6akp' size='340' side='right'caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>
	== Structural highlights ==		== Structural highlights ==
-	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6AKP FirstGlance]. <br>	+	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure with sequence from [http://en.wikipedia.org/wiki/ ] and [http://en.wikipedia.org/wiki/Human Human]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6AKP FirstGlance]. <br>
	</td></tr><tr id='ligand'><td class="sblockLbl"><b>[[Ligand\|Ligands:]]</b></td><td class="sblockDat"><scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene></td></tr>		</td></tr><tr id='ligand'><td class="sblockLbl"><b>[[Ligand\|Ligands:]]</b></td><td class="sblockDat"><scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene></td></tr>
		+	<tr id='gene'><td class="sblockLbl"><b>[[Gene\|Gene:]]</b></td><td class="sblockDat">FOXC2, FKHL14, MFH1 ([http://www.ncbi.nlm.nih.gov/Taxonomy/Browser/wwwtax.cgi?mode=Info&srchmode=5&id=9606 HUMAN])</td></tr>
	<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [http://pdbe.org/6akp PDBe], [http://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [http://www.ebi.ac.uk/pdbsum/6akp PDBsum], [http://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>		<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [http://pdbe.org/6akp PDBe], [http://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [http://www.ebi.ac.uk/pdbsum/6akp PDBsum], [http://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>
	</table>		</table>
Line 24:		Line 25:
	__TOC__		__TOC__
	</StructureSection>		</StructureSection>
		+	[[Category: Human]]
		+	[[Category: Large Structures]]
	[[Category: Chen, X]]		[[Category: Chen, X]]
	[[Category: Chen, Y]]		[[Category: Chen, Y]]

OCA at 09:43, 13 February 2019

OCA — Wed, 13 Feb 2019 09:43:38 GMT

←Older revision		Revision as of 09:43, 13 February 2019
Line 1:		Line 1:
-	'''Unreleased structure'''

-	~~The entry~~ 6akp is ~~ON HOLD~~ ~~until Paper~~ Publication	+	==Crystal Structural of FOXC2 DNA binding domain bound to PC promoter==
		+	<StructureSection load='6akp' size='340' side='right' caption='[[6akp]], [[Resolution\|resolution]] 2.32Å' scene=''>
		+	== Structural highlights ==
		+	<table><tr><td colspan='2'>[[6akp]] is a 3 chain structure. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=6AKP OCA]. For a <b>guided tour on the structure components</b> use [http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6AKP FirstGlance]. <br>
		+	</td></tr><tr id='ligand'><td class="sblockLbl"><b>[[Ligand\|Ligands:]]</b></td><td class="sblockDat"><scene name='pdbligand=MG:MAGNESIUM+ION'>MG</scene></td></tr>
		+	<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=6akp FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=6akp OCA], [http://pdbe.org/6akp PDBe], [http://www.rcsb.org/pdb/explore.do?structureId=6akp RCSB], [http://www.ebi.ac.uk/pdbsum/6akp PDBsum], [http://prosat.h-its.org/prosat/prosatexe?pdbcode=6akp ProSAT]</span></td></tr>
		+	</table>
		+	== Disease ==
		+	[[http://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Defects in FOXC2 are the cause of lymphedema hereditary type 2 (LMPH2) [MIM:[http://omim.org/entry/153200 153200]]; also known as Meige lymphedema. Hereditary lymphedema is a chronic disabling condition which results in swelling of the extremities due to altered lymphatic flow. Patients with lymphedema suffer from recurrent local infections, and physical impairment.<ref>PMID:11078474</ref> Defects in FOXC2 are a cause of lymphedema-yellow nails (LYYN) [MIM:[http://omim.org/entry/153300 153300]]. LYYN is characterized by yellow, dystrophic, thick and slowly growing nails, associated with lymphedema and respiratory involvement. Lymphedema occurs more often in the lower limbs. It can appear at birth or later in life. Onset generally follows the onset of ungual abnormalities. Defects in FOXC2 are a cause of lymphedema-distichiasis (LYD) [MIM:[http://omim.org/entry/153400 153400]]. LYD is characterized by primary limb lymphedema usually starting at puberty (but in some cases later or at birth) and associated with distichiasis (double rows of eyelashes, with extra eyelashes growing from the Meibomian gland orifices).<ref>PMID:11499682</ref>
		+	== Function ==
		+	[[http://www.uniprot.org/uniprot/FOXC2_HUMAN FOXC2_HUMAN]] Transcriptional activator. Might be involved in the formation of special mesenchymal tissues.<ref>PMID:9169153</ref>
		+	<div style="background-color:#fffaf0;">
		+	== Publication Abstract from PubMed ==
		+	The FOXC family of transcription factors (FOXC1 and FOXC2) plays essential roles in the regulation of embryonic, ocular, and cardiac development. Mutations and abnormal expression of FOXC proteins are implicated in genetic diseases as well as cancer. In this study, we determined two crystal structures of the DNA-binding domain (DBD) of human FOXC2 protein, in complex with different DNA sites. The FOXC2-DBD adopts the winged-helix fold with helix H3 contributing to all the base specific contacts, while the N-terminus, wing 1, and the C-terminus of FOXC2-DBD all make additional contacts with the phosphate groups of DNA. Our structural, biochemical, and bioinformatics analyses allow us to revise the previously proposed DNA recognition mechanism and provide a model of DNA binding for the FOXC proteins. In addition, our structural analysis and accompanying biochemical assays provide a molecular basis for understanding disease-causing mutations in FOXC1 and FOXC2.

-	~~Authors:~~ Chen, X., Wei, H., Li, J., Liang, X., Dai, S., Jiang, L., Guo, M., Chen, Y.	+	Structural basis for DNA recognition by FOXC2.,Chen X, Wei H, Li J, Liang X, Dai S, Jiang L, Guo M, Qu L, Chen Z, Chen L, Chen Y Nucleic Acids Res. 2019 Feb 5. pii: 5306577. doi: 10.1093/nar/gkz077. PMID:30722065<ref>PMID:30722065</ref>

-	~~Description: Crystal Structural~~ of ~~FOXC2 DNA binding domain bound to PC promoter~~	+	From MEDLINE®/PubMed®, a database of the U.S. National Library of Medicine.<br>
-	~~[[Category: Unreleased Structures]]~~	+	</div>
-	~~[[Category~~: ~~Guo, M]]~~	+	<div class="pdbe-citations 6akp" style="background-color:#fffaf0;"></div>
-	~~[[Category: Liang, X]]~~	+	== References ==
-	~~[[Category: Jiang, L]]~~	+	<references/>
		+	__TOC__
		+	</StructureSection>
	[[Category: Chen, X]]		[[Category: Chen, X]]
		+	[[Category: Chen, Y]]
	[[Category: Dai, S]]		[[Category: Dai, S]]
		+	[[Category: Guo, M]]
		+	[[Category: Jiang, L]]
	[[Category: Li, J]]		[[Category: Li, J]]
		+	[[Category: Liang, X]]
	[[Category: Wei, H]]		[[Category: Wei, H]]
-	[[Category: ~~Chen, Y~~]]	+	[[Category: Crystal structural]]
		+	[[Category: Dna binding domain]]
		+	[[Category: Dna binding protein-dna complex]]
		+	[[Category: Dna recognition]]
		+	[[Category: Foxc]]
		+	[[Category: Lymphoedema distichiasis syndrome]]

OCA at 06:08, 10 October 2018

OCA — Wed, 10 Oct 2018 06:08:12 GMT

←Older revision		Revision as of 06:08, 10 October 2018
Line 1:		Line 1:
	'''Unreleased structure'''		'''Unreleased structure'''

-	The entry 6akp is ON HOLD	+	The entry 6akp is ON HOLD until Paper Publication

	Authors: Chen, X., Wei, H., Li, J., Liang, X., Dai, S., Jiang, L., Guo, M., Chen, Y.		Authors: Chen, X., Wei, H., Li, J., Liang, X., Dai, S., Jiang, L., Guo, M., Chen, Y.

OCA: Protected "6akp" [edit=sysop:move=sysop]

OCA — Wed, 12 Sep 2018 05:55:45 GMT

Protected "6akp" [edit=sysop:move=sysop]

←Older revision

Revision as of 05:55, 12 September 2018

OCA: New page: '''Unreleased structure''' The entry 6akp is ON HOLD Authors: Chen, X., Wei, H., Li, J., Liang, X., Dai, S., Jiang, L., Guo, M., Chen, Y. Description: Crystal Structural of FOXC2 DNA b...

OCA — Wed, 12 Sep 2018 05:55:43 GMT

New page: '''Unreleased structure''' The entry 6akp is ON HOLD Authors: Chen, X., Wei, H., Li, J., Liang, X., Dai, S., Jiang, L., Guo, M., Chen, Y. Description: Crystal Structural of FOXC2 DNA b...

New page

'''Unreleased structure'''

The entry 6akp is ON HOLD

Authors: Chen, X., Wei, H., Li, J., Liang, X., Dai, S., Jiang, L., Guo, M., Chen, Y.

Description: Crystal Structural of FOXC2 DNA binding domain bound to PC promoter
[[Category: Unreleased Structures]]
[[Category: Guo, M]]
[[Category: Liang, X]]
[[Category: Jiang, L]]
[[Category: Chen, X]]
[[Category: Dai, S]]
[[Category: Li, J]]
[[Category: Wei, H]]
[[Category: Chen, Y]]