1bym

From Proteopedia

(Difference between revisions)
Jump to: navigation, search
(New page: 200px<br /><applet load="1bym" size="450" color="white" frame="true" align="right" spinBox="true" caption="1bym" /> '''SOLUTION STRUCTURES OF THE C-TERMINAL DOMAIN...)
Line 1: Line 1:
-
[[Image:1bym.jpg|left|200px]]<br /><applet load="1bym" size="450" color="white" frame="true" align="right" spinBox="true"
+
[[Image:1bym.jpg|left|200px]]<br /><applet load="1bym" size="350" color="white" frame="true" align="right" spinBox="true"
caption="1bym" />
caption="1bym" />
'''SOLUTION STRUCTURES OF THE C-TERMINAL DOMAIN OF DIPHTHERIA TOXIN REPRESSOR'''<br />
'''SOLUTION STRUCTURES OF THE C-TERMINAL DOMAIN OF DIPHTHERIA TOXIN REPRESSOR'''<br />
==Overview==
==Overview==
-
The diphtheria toxin repressor (DtxR) is the best-characterized member of, a family of homologous proteins that regulate iron uptake and virulence, gene expression in the Gram-positive bacteria. DtxR contains two domains, that are separated by a short, unstructured linker. The N-terminal domain, is structurally well-defined and is responsible for Fe2+ binding, dimerization, and DNA binding. The C-terminal domain adopts a fold similar, to eukaryotic Src homology 3 domains, but the functional role of the, C-terminal domain in repressor activity is unknown. The solution structure, of the C-terminal domain, consisting of residues N130-L226 plus a, 13-residue N-terminal extension, has been determined by using NMR, spectroscopy. Residues before A147 are highly mobile and adopt a random, coil conformation, but residues A147-L226 form a single structured domain, consisting of five beta-strands and three helices arranged into a, partially orthogonal, two-sheet beta-barrel, similar to the structure, observed in the crystalline Co2+ complex of full-length DtxR. Chemical, shift perturbation studies demonstrate that a proline-rich peptide, corresponding to residues R125-G139 of intact DtxR binds to the C-terminal, domain in a pocket formed by residues in beta-strands 2, 3, and 5, and, helix 3. Binding of the proline-rich peptide by the C-terminal domain of, DtxR presents an example of peptide binding by a prokaryotic Src homology, 3-like protein. The results of this study, combined with previous x-ray, studies of intact DtxR, provide insights into a possible biological, function of the C-terminal domain in regulating repressor activity.
+
The diphtheria toxin repressor (DtxR) is the best-characterized member of a family of homologous proteins that regulate iron uptake and virulence gene expression in the Gram-positive bacteria. DtxR contains two domains that are separated by a short, unstructured linker. The N-terminal domain is structurally well-defined and is responsible for Fe2+ binding, dimerization, and DNA binding. The C-terminal domain adopts a fold similar to eukaryotic Src homology 3 domains, but the functional role of the C-terminal domain in repressor activity is unknown. The solution structure of the C-terminal domain, consisting of residues N130-L226 plus a 13-residue N-terminal extension, has been determined by using NMR spectroscopy. Residues before A147 are highly mobile and adopt a random coil conformation, but residues A147-L226 form a single structured domain consisting of five beta-strands and three helices arranged into a partially orthogonal, two-sheet beta-barrel, similar to the structure observed in the crystalline Co2+ complex of full-length DtxR. Chemical shift perturbation studies demonstrate that a proline-rich peptide corresponding to residues R125-G139 of intact DtxR binds to the C-terminal domain in a pocket formed by residues in beta-strands 2, 3, and 5, and helix 3. Binding of the proline-rich peptide by the C-terminal domain of DtxR presents an example of peptide binding by a prokaryotic Src homology 3-like protein. The results of this study, combined with previous x-ray studies of intact DtxR, provide insights into a possible biological function of the C-terminal domain in regulating repressor activity.
==About this Structure==
==About this Structure==
-
1BYM is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Corynebacterium_diphtheriae Corynebacterium diphtheriae]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1BYM OCA].
+
1BYM is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Corynebacterium_diphtheriae Corynebacterium diphtheriae]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1BYM OCA].
==Reference==
==Reference==
Line 13: Line 13:
[[Category: Corynebacterium diphtheriae]]
[[Category: Corynebacterium diphtheriae]]
[[Category: Single protein]]
[[Category: Single protein]]
-
[[Category: Caspar, D.L.D.]]
+
[[Category: Caspar, D L.D.]]
-
[[Category: Logan, T.M.]]
+
[[Category: Logan, T M.]]
-
[[Category: Murphy, J.R.]]
+
[[Category: Murphy, J R.]]
-
[[Category: Twigg, P.D.]]
+
[[Category: Twigg, P D.]]
[[Category: Wang, G.]]
[[Category: Wang, G.]]
-
[[Category: Wylie, G.P.]]
+
[[Category: Wylie, G P.]]
[[Category: c-terminal domain]]
[[Category: c-terminal domain]]
[[Category: dtxr]]
[[Category: dtxr]]
Line 26: Line 26:
[[Category: transcription regulation]]
[[Category: transcription regulation]]
-
''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Tue Nov 20 12:01:54 2007''
+
''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 12:00:32 2008''

Revision as of 10:00, 21 February 2008


1bym

Drag the structure with the mouse to rotate

SOLUTION STRUCTURES OF THE C-TERMINAL DOMAIN OF DIPHTHERIA TOXIN REPRESSOR

Overview

The diphtheria toxin repressor (DtxR) is the best-characterized member of a family of homologous proteins that regulate iron uptake and virulence gene expression in the Gram-positive bacteria. DtxR contains two domains that are separated by a short, unstructured linker. The N-terminal domain is structurally well-defined and is responsible for Fe2+ binding, dimerization, and DNA binding. The C-terminal domain adopts a fold similar to eukaryotic Src homology 3 domains, but the functional role of the C-terminal domain in repressor activity is unknown. The solution structure of the C-terminal domain, consisting of residues N130-L226 plus a 13-residue N-terminal extension, has been determined by using NMR spectroscopy. Residues before A147 are highly mobile and adopt a random coil conformation, but residues A147-L226 form a single structured domain consisting of five beta-strands and three helices arranged into a partially orthogonal, two-sheet beta-barrel, similar to the structure observed in the crystalline Co2+ complex of full-length DtxR. Chemical shift perturbation studies demonstrate that a proline-rich peptide corresponding to residues R125-G139 of intact DtxR binds to the C-terminal domain in a pocket formed by residues in beta-strands 2, 3, and 5, and helix 3. Binding of the proline-rich peptide by the C-terminal domain of DtxR presents an example of peptide binding by a prokaryotic Src homology 3-like protein. The results of this study, combined with previous x-ray studies of intact DtxR, provide insights into a possible biological function of the C-terminal domain in regulating repressor activity.

About this Structure

1BYM is a Single protein structure of sequence from Corynebacterium diphtheriae. Full crystallographic information is available from OCA.

Reference

Solution structure and peptide binding studies of the C-terminal src homology 3-like domain of the diphtheria toxin repressor protein., Wang G, Wylie GP, Twigg PD, Caspar DL, Murphy JR, Logan TM, Proc Natl Acad Sci U S A. 1999 May 25;96(11):6119-24. PMID:10339551

Page seeded by OCA on Thu Feb 21 12:00:32 2008

Proteopedia Page Contributors and Editors (what is this?)

OCA

Personal tools