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1l9m

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(New page: 200px<br /> <applet load="1l9m" size="450" color="white" frame="true" align="right" spinBox="true" caption="1l9m, resolution 2.10&Aring;" /> '''Three-dimensional s...)
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'''Three-dimensional structure of the human transglutaminase 3 enzyme: binding of calcium ions change structure for activation'''<br />
'''Three-dimensional structure of the human transglutaminase 3 enzyme: binding of calcium ions change structure for activation'''<br />
==Overview==
==Overview==
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Transglutaminase (TGase) enzymes catalyze the formation of covalent, cross-links between protein-bound glutamines and lysines in a, calcium-dependent manner, but the role of Ca(2+) ions remains unclear. The, TGase 3 isoform is widely expressed and is important for epithelial, barrier formation. It is a zymogen, requiring proteolysis for activity. We, have solved the three-dimensional structures of the zymogen and the, activated forms at 2.2 and 2.1 A resolution, respectively, and examined, the role of Ca(2+) ions. The zymogen binds one ion tightly that cannot be, exchanged. Upon proteolysis, the enzyme exothermally acquires two more, Ca(2+) ions that activate the enzyme, are exchangeable and are, functionally replaceable by other lanthanide trivalent cations. Binding of, a Ca(2+) ion at one of these sites opens a channel which exposes the key, Trp236 and Trp327 residues that control substrate access to the active, site. Together, these biochemical and structural data reveal for the first, time in a TGase enzyme that Ca(2+) ions induce structural changes which at, least in part dictate activity and, moreover, may confer substrate, specificity.
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Transglutaminase (TGase) enzymes catalyze the formation of covalent cross-links between protein-bound glutamines and lysines in a calcium-dependent manner, but the role of Ca(2+) ions remains unclear. The TGase 3 isoform is widely expressed and is important for epithelial barrier formation. It is a zymogen, requiring proteolysis for activity. We have solved the three-dimensional structures of the zymogen and the activated forms at 2.2 and 2.1 A resolution, respectively, and examined the role of Ca(2+) ions. The zymogen binds one ion tightly that cannot be exchanged. Upon proteolysis, the enzyme exothermally acquires two more Ca(2+) ions that activate the enzyme, are exchangeable and are functionally replaceable by other lanthanide trivalent cations. Binding of a Ca(2+) ion at one of these sites opens a channel which exposes the key Trp236 and Trp327 residues that control substrate access to the active site. Together, these biochemical and structural data reveal for the first time in a TGase enzyme that Ca(2+) ions induce structural changes which at least in part dictate activity and, moreover, may confer substrate specificity.
==About this Structure==
==About this Structure==
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1L9M is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with BR, CL and CA as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/Protein-glutamine_gamma-glutamyltransferase Protein-glutamine gamma-glutamyltransferase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=2.3.2.13 2.3.2.13] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1L9M OCA].
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1L9M is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=BR:'>BR</scene>, <scene name='pdbligand=CL:'>CL</scene> and <scene name='pdbligand=CA:'>CA</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/Protein-glutamine_gamma-glutamyltransferase Protein-glutamine gamma-glutamyltransferase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=2.3.2.13 2.3.2.13] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1L9M OCA].
==Reference==
==Reference==
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[[Category: x-ray structure]]
[[Category: x-ray structure]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 17:58:03 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 13:42:50 2008''

Revision as of 11:42, 21 February 2008


1l9m, resolution 2.10Å

Drag the structure with the mouse to rotate

Three-dimensional structure of the human transglutaminase 3 enzyme: binding of calcium ions change structure for activation

Overview

Transglutaminase (TGase) enzymes catalyze the formation of covalent cross-links between protein-bound glutamines and lysines in a calcium-dependent manner, but the role of Ca(2+) ions remains unclear. The TGase 3 isoform is widely expressed and is important for epithelial barrier formation. It is a zymogen, requiring proteolysis for activity. We have solved the three-dimensional structures of the zymogen and the activated forms at 2.2 and 2.1 A resolution, respectively, and examined the role of Ca(2+) ions. The zymogen binds one ion tightly that cannot be exchanged. Upon proteolysis, the enzyme exothermally acquires two more Ca(2+) ions that activate the enzyme, are exchangeable and are functionally replaceable by other lanthanide trivalent cations. Binding of a Ca(2+) ion at one of these sites opens a channel which exposes the key Trp236 and Trp327 residues that control substrate access to the active site. Together, these biochemical and structural data reveal for the first time in a TGase enzyme that Ca(2+) ions induce structural changes which at least in part dictate activity and, moreover, may confer substrate specificity.

About this Structure

1L9M is a Single protein structure of sequence from Homo sapiens with , and as ligands. Active as Protein-glutamine gamma-glutamyltransferase, with EC number 2.3.2.13 Full crystallographic information is available from OCA.

Reference

Three-dimensional structure of the human transglutaminase 3 enzyme: binding of calcium ions changes structure for activation., Ahvazi B, Kim HC, Kee SH, Nemes Z, Steinert PM, EMBO J. 2002 May 1;21(9):2055-67. PMID:11980702

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