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1mo7

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(New page: 200px<br /><applet load="1mo7" size="450" color="white" frame="true" align="right" spinBox="true" caption="1mo7" /> '''ATPase'''<br /> ==Overview== The Na,K-ATPas...)
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'''ATPase'''<br />
'''ATPase'''<br />
==Overview==
==Overview==
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The Na,K-ATPase hydrolyzes ATP to drive the coupled extrusion and uptake, of Na+ and K+ ions across the plasma membrane. Here, we report two, high-resolution NMR structures of the 213-residue nucleotide-binding, domain of rat alpha1 Na,K-ATPase, determined in the absence and the, presence of ATP. The nucleotide binds in the anti conformation and shows a, relative paucity of interactions with the protein, reflecting the, low-affinity ATP-binding state. Binding of ATP induces substantial, conformational changes in the binding pocket and in residues located in, the hinge region connecting the N- and P-domains. Structural comparison, with the Ca-ATPase stabilized by the inhibitor thapsigargin, E2(TG), and, the model of the H-ATPase in the E1 form suggests that the observed, changes may trigger the series of events necessary for the release of the, K+ ions and/or disengagement of the A-domain, leading to the eventual, transfer of the gamma-phosphate group to the invariant Asp369.
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The Na,K-ATPase hydrolyzes ATP to drive the coupled extrusion and uptake of Na+ and K+ ions across the plasma membrane. Here, we report two high-resolution NMR structures of the 213-residue nucleotide-binding domain of rat alpha1 Na,K-ATPase, determined in the absence and the presence of ATP. The nucleotide binds in the anti conformation and shows a relative paucity of interactions with the protein, reflecting the low-affinity ATP-binding state. Binding of ATP induces substantial conformational changes in the binding pocket and in residues located in the hinge region connecting the N- and P-domains. Structural comparison with the Ca-ATPase stabilized by the inhibitor thapsigargin, E2(TG), and the model of the H-ATPase in the E1 form suggests that the observed changes may trigger the series of events necessary for the release of the K+ ions and/or disengagement of the A-domain, leading to the eventual transfer of the gamma-phosphate group to the invariant Asp369.
==About this Structure==
==About this Structure==
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1MO7 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Rattus_norvegicus Rattus norvegicus]. Active as [http://en.wikipedia.org/wiki/Sodium/potassium-exchanging_ATPase Sodium/potassium-exchanging ATPase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.6.3.9 3.6.3.9] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1MO7 OCA].
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1MO7 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Rattus_norvegicus Rattus norvegicus]. Active as [http://en.wikipedia.org/wiki/Sodium/potassium-exchanging_ATPase Sodium/potassium-exchanging ATPase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.6.3.9 3.6.3.9] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1MO7 OCA].
==Reference==
==Reference==
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[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Sodium/potassium-exchanging ATPase]]
[[Category: Sodium/potassium-exchanging ATPase]]
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[[Category: Abrahams, J.P.]]
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[[Category: Abrahams, J P.]]
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[[Category: Gloor, S.M.]]
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[[Category: Gloor, S M.]]
[[Category: Guentert, P.]]
[[Category: Guentert, P.]]
[[Category: Hilge, M.]]
[[Category: Hilge, M.]]
[[Category: Siegal, G.]]
[[Category: Siegal, G.]]
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[[Category: Vuister, G.W.]]
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[[Category: Vuister, G W.]]
[[Category: six-stranded]]
[[Category: six-stranded]]
[[Category: twisted beta sheet]]
[[Category: twisted beta sheet]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Tue Nov 20 21:34:38 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 13:57:13 2008''

Revision as of 11:57, 21 February 2008


1mo7

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ATPase

Overview

The Na,K-ATPase hydrolyzes ATP to drive the coupled extrusion and uptake of Na+ and K+ ions across the plasma membrane. Here, we report two high-resolution NMR structures of the 213-residue nucleotide-binding domain of rat alpha1 Na,K-ATPase, determined in the absence and the presence of ATP. The nucleotide binds in the anti conformation and shows a relative paucity of interactions with the protein, reflecting the low-affinity ATP-binding state. Binding of ATP induces substantial conformational changes in the binding pocket and in residues located in the hinge region connecting the N- and P-domains. Structural comparison with the Ca-ATPase stabilized by the inhibitor thapsigargin, E2(TG), and the model of the H-ATPase in the E1 form suggests that the observed changes may trigger the series of events necessary for the release of the K+ ions and/or disengagement of the A-domain, leading to the eventual transfer of the gamma-phosphate group to the invariant Asp369.

About this Structure

1MO7 is a Single protein structure of sequence from Rattus norvegicus. Active as Sodium/potassium-exchanging ATPase, with EC number 3.6.3.9 Full crystallographic information is available from OCA.

Reference

ATP-induced conformational changes of the nucleotide-binding domain of Na,K-ATPase., Hilge M, Siegal G, Vuister GW, Guntert P, Gloor SM, Abrahams JP, Nat Struct Biol. 2003 Jun;10(6):468-74. PMID:12730684

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