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1r1x

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[[Image:1r1x.gif|left|200px]]<br />
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[[Image:1r1x.gif|left|200px]]<br /><applet load="1r1x" size="350" color="white" frame="true" align="right" spinBox="true"
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<applet load="1r1x" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="1r1x, resolution 2.15&Aring;" />
caption="1r1x, resolution 2.15&Aring;" />
'''Crystal structure of oxy-human hemoglobin Bassett at 2.15 angstrom'''<br />
'''Crystal structure of oxy-human hemoglobin Bassett at 2.15 angstrom'''<br />
==Overview==
==Overview==
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Hemoglobin (Hb) Bassett, an abnormal Hb variant with a markedly reduced, oxygen affinity, was discovered in a Caucasian (Anglo-Saxon) male child, who experienced episodes of cyanosis. Cation-exchange and reversed-phase, (RP) high-performance liquid chromatography (HPLC) showed that the patient, has an abnormal Hb, with a mutation in the alpha-globin. Tryptic peptide, digest of the abnormal alpha-globin with subsequent HPLC analysis revealed, abnormal elution of the alpha-T11 peptide. Further studies with Edman, sequencing and electrospray mass spectrometry of tryptic peptide, alpha-T11, as well as structural analysis by X-ray crystallography, revealed an Asp--&gt;Ala substitution at the alpha94 (G1) position, a match, for Hb Bassett. Detailed functional studies showed that this Hb variant, had a markedly reduced oxygen affinity (P(50) at pH 7.0 = 22 mmHg; Hb A, P(50) = 10.5 mmHg), reduced Bohr effect (-0.26 compared to - 0.54 in Hb, A), and low subunit cooperativity (n = 1.4, compared to 2.6 in Hb A)., X-ray crystallography results explain the probable effects of the, structural modification on the oxygen-binding properties of this Hb, variant.
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Hemoglobin (Hb) Bassett, an abnormal Hb variant with a markedly reduced oxygen affinity, was discovered in a Caucasian (Anglo-Saxon) male child who experienced episodes of cyanosis. Cation-exchange and reversed-phase (RP) high-performance liquid chromatography (HPLC) showed that the patient has an abnormal Hb, with a mutation in the alpha-globin. Tryptic peptide digest of the abnormal alpha-globin with subsequent HPLC analysis revealed abnormal elution of the alpha-T11 peptide. Further studies with Edman sequencing and electrospray mass spectrometry of tryptic peptide alpha-T11, as well as structural analysis by X-ray crystallography revealed an Asp--&gt;Ala substitution at the alpha94 (G1) position, a match for Hb Bassett. Detailed functional studies showed that this Hb variant had a markedly reduced oxygen affinity (P(50) at pH 7.0 = 22 mmHg; Hb A P(50) = 10.5 mmHg), reduced Bohr effect (-0.26 compared to - 0.54 in Hb A), and low subunit cooperativity (n = 1.4, compared to 2.6 in Hb A). X-ray crystallography results explain the probable effects of the structural modification on the oxygen-binding properties of this Hb variant.
==Disease==
==Disease==
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==About this Structure==
==About this Structure==
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1R1X is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with CMO, HEM and MBN as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1R1X OCA].
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1R1X is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=CMO:'>CMO</scene>, <scene name='pdbligand=HEM:'>HEM</scene> and <scene name='pdbligand=MBN:'>MBN</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1R1X OCA].
==Reference==
==Reference==
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[[Category: Protein complex]]
[[Category: Protein complex]]
[[Category: Abdulmalik, O.]]
[[Category: Abdulmalik, O.]]
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[[Category: Abraham, D.J.]]
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[[Category: Abraham, D J.]]
[[Category: Asakura, T.]]
[[Category: Asakura, T.]]
[[Category: Dhaikin, Y.]]
[[Category: Dhaikin, Y.]]
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[[Category: Lerner, N.B.]]
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[[Category: Lerner, N B.]]
[[Category: Ochotorena, J.]]
[[Category: Ochotorena, J.]]
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[[Category: Safo, M.K.]]
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[[Category: Safo, M K.]]
[[Category: CMO]]
[[Category: CMO]]
[[Category: HEM]]
[[Category: HEM]]
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[[Category: rochester]]
[[Category: rochester]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 18:58:36 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 14:46:08 2008''

Revision as of 12:46, 21 February 2008


1r1x, resolution 2.15Å

Drag the structure with the mouse to rotate

Crystal structure of oxy-human hemoglobin Bassett at 2.15 angstrom

Contents

Overview

Hemoglobin (Hb) Bassett, an abnormal Hb variant with a markedly reduced oxygen affinity, was discovered in a Caucasian (Anglo-Saxon) male child who experienced episodes of cyanosis. Cation-exchange and reversed-phase (RP) high-performance liquid chromatography (HPLC) showed that the patient has an abnormal Hb, with a mutation in the alpha-globin. Tryptic peptide digest of the abnormal alpha-globin with subsequent HPLC analysis revealed abnormal elution of the alpha-T11 peptide. Further studies with Edman sequencing and electrospray mass spectrometry of tryptic peptide alpha-T11, as well as structural analysis by X-ray crystallography revealed an Asp-->Ala substitution at the alpha94 (G1) position, a match for Hb Bassett. Detailed functional studies showed that this Hb variant had a markedly reduced oxygen affinity (P(50) at pH 7.0 = 22 mmHg; Hb A P(50) = 10.5 mmHg), reduced Bohr effect (-0.26 compared to - 0.54 in Hb A), and low subunit cooperativity (n = 1.4, compared to 2.6 in Hb A). X-ray crystallography results explain the probable effects of the structural modification on the oxygen-binding properties of this Hb variant.

Disease

Known diseases associated with this structure: Erythremias, alpha- OMIM:[141800], Erythremias, beta- OMIM:[141900], Erythrocytosis OMIM:[141850], HPFH, deletion type OMIM:[141900], Heinz body anemia OMIM:[141850], Heinz body anemias, alpha- OMIM:[141800], Heinz body anemias, beta- OMIM:[141900], Hemoglobin H disease OMIM:[141850], Hypochromic microcytic anemia OMIM:[141850], Methemoglobinemias, alpha- OMIM:[141800], Methemoglobinemias, beta- OMIM:[141900], Sickle cell anemia OMIM:[141900], Thalassemia, alpha- OMIM:[141850], Thalassemia-beta, dominant inclusion-body OMIM:[141900], Thalassemias, alpha- OMIM:[141800], Thalassemias, beta- OMIM:[141900]

About this Structure

1R1X is a Protein complex structure of sequences from Homo sapiens with , and as ligands. Full crystallographic information is available from OCA.

Reference

Characterization of hemoglobin bassett (alpha94Asp-->Ala), a variant with very low oxygen affinity., Abdulmalik O, Safo MK, Lerner NB, Ochotorena J, Daikhin E, Lakka V, Santacroce R, Abraham DJ, Asakura T, Am J Hematol. 2004 Nov;77(3):268-76. PMID:15495251

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