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1yyz

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(New page: 200px<br /><applet load="1yyz" size="450" color="white" frame="true" align="right" spinBox="true" caption="1yyz, resolution 1.85&Aring;" /> '''R-State AMP Complex ...)
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'''R-State AMP Complex Reveals Initial Steps of the Quaternary Transition of Fructose-1,6-bisphosphatase'''<br />
'''R-State AMP Complex Reveals Initial Steps of the Quaternary Transition of Fructose-1,6-bisphosphatase'''<br />
==Overview==
==Overview==
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AMP transforms fructose-1,6-bisphosphatase from its active R-state to its, inactive T-state; however, the mechanism of that transformation is poorly, understood. The mutation of Ala(54) to leucine destabilizes the T-state of, fructose-1,6-bisphosphatase. The mutant enzyme retains wild-type levels of, activity, but the concentration of AMP that causes 50% inhibition, increases 50-fold. In the absence of AMP, the Leu(54) enzyme adopts an, R-state conformation nearly identical to that of the wild-type enzyme. The, mutant enzyme, however, grows in two crystal forms in the presence of, saturating AMP. In one form, the AMP-bound tetramer is in a T-like, conformation, whereas in the other form, the AMP-bound tetramer is in a, R-like conformation. The latter reveals conformational changes in two, helices due to the binding of AMP. Helix H1 moves toward the center of the, tetramer and displaces Ile(10) from a hydrophobic pocket. The displacement, of Ile(10) exposes a hydrophobic surface critical to interactions that, stabilize the T-state. Helix H2 moves away from the center of the, tetramer, breaking hydrogen bonds with a buried loop (residues 187-195) in, an adjacent subunit. The same hydrogen bonds reform but only after the, quaternary transition to the T-state. Proposed here is a model that, accounts for the quaternary transition and cooperativity in the inhibition, of catalysis by AMP.
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AMP transforms fructose-1,6-bisphosphatase from its active R-state to its inactive T-state; however, the mechanism of that transformation is poorly understood. The mutation of Ala(54) to leucine destabilizes the T-state of fructose-1,6-bisphosphatase. The mutant enzyme retains wild-type levels of activity, but the concentration of AMP that causes 50% inhibition increases 50-fold. In the absence of AMP, the Leu(54) enzyme adopts an R-state conformation nearly identical to that of the wild-type enzyme. The mutant enzyme, however, grows in two crystal forms in the presence of saturating AMP. In one form, the AMP-bound tetramer is in a T-like conformation, whereas in the other form, the AMP-bound tetramer is in a R-like conformation. The latter reveals conformational changes in two helices due to the binding of AMP. Helix H1 moves toward the center of the tetramer and displaces Ile(10) from a hydrophobic pocket. The displacement of Ile(10) exposes a hydrophobic surface critical to interactions that stabilize the T-state. Helix H2 moves away from the center of the tetramer, breaking hydrogen bonds with a buried loop (residues 187-195) in an adjacent subunit. The same hydrogen bonds reform but only after the quaternary transition to the T-state. Proposed here is a model that accounts for the quaternary transition and cooperativity in the inhibition of catalysis by AMP.
==About this Structure==
==About this Structure==
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1YYZ is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Sus_scrofa Sus scrofa] with F6P, PO4, MG and AMP as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/Fructose-bisphosphatase Fructose-bisphosphatase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.1.3.11 3.1.3.11] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1YYZ OCA].
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1YYZ is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Sus_scrofa Sus scrofa] with <scene name='pdbligand=F6P:'>F6P</scene>, <scene name='pdbligand=PO4:'>PO4</scene>, <scene name='pdbligand=MG:'>MG</scene> and <scene name='pdbligand=AMP:'>AMP</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/Fructose-bisphosphatase Fructose-bisphosphatase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.1.3.11 3.1.3.11] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1YYZ OCA].
==Reference==
==Reference==
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[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Sus scrofa]]
[[Category: Sus scrofa]]
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[[Category: Fromm, H.J.]]
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[[Category: Fromm, H J.]]
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[[Category: Honzatko, R.B.]]
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[[Category: Honzatko, R B.]]
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[[Category: Iancu, C.V.]]
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[[Category: Iancu, C V.]]
[[Category: Mukund, S.]]
[[Category: Mukund, S.]]
[[Category: AMP]]
[[Category: AMP]]
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[[Category: intermediate states]]
[[Category: intermediate states]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 16:10:41 2008''

Revision as of 14:10, 21 February 2008


1yyz, resolution 1.85Å

Drag the structure with the mouse to rotate

R-State AMP Complex Reveals Initial Steps of the Quaternary Transition of Fructose-1,6-bisphosphatase

Overview

AMP transforms fructose-1,6-bisphosphatase from its active R-state to its inactive T-state; however, the mechanism of that transformation is poorly understood. The mutation of Ala(54) to leucine destabilizes the T-state of fructose-1,6-bisphosphatase. The mutant enzyme retains wild-type levels of activity, but the concentration of AMP that causes 50% inhibition increases 50-fold. In the absence of AMP, the Leu(54) enzyme adopts an R-state conformation nearly identical to that of the wild-type enzyme. The mutant enzyme, however, grows in two crystal forms in the presence of saturating AMP. In one form, the AMP-bound tetramer is in a T-like conformation, whereas in the other form, the AMP-bound tetramer is in a R-like conformation. The latter reveals conformational changes in two helices due to the binding of AMP. Helix H1 moves toward the center of the tetramer and displaces Ile(10) from a hydrophobic pocket. The displacement of Ile(10) exposes a hydrophobic surface critical to interactions that stabilize the T-state. Helix H2 moves away from the center of the tetramer, breaking hydrogen bonds with a buried loop (residues 187-195) in an adjacent subunit. The same hydrogen bonds reform but only after the quaternary transition to the T-state. Proposed here is a model that accounts for the quaternary transition and cooperativity in the inhibition of catalysis by AMP.

About this Structure

1YYZ is a Single protein structure of sequence from Sus scrofa with , , and as ligands. Active as Fructose-bisphosphatase, with EC number 3.1.3.11 Full crystallographic information is available from OCA.

Reference

R-state AMP complex reveals initial steps of the quaternary transition of fructose-1,6-bisphosphatase., Iancu CV, Mukund S, Fromm HJ, Honzatko RB, J Biol Chem. 2005 May 20;280(20):19737-45. Epub 2005 Mar 14. PMID:15767255

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