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3bix

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==Overview==
==Overview==
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Neurexins and neuroligins provide trans-synaptic connectivity by the, Ca(2+)-dependent interaction of their alternatively spliced extracellular, domains. Neuroligins specify synapses in an activity-dependent manner, presumably by binding to neurexins. Here, we present the crystal, structures of neuroligin-1 in isolation and in complex with, neurexin-1beta. Neuroligin-1 forms a constitutive dimer, and two, neurexin-1beta monomers bind to two identical surfaces on the opposite, faces of the neuroligin-1 dimer to form a heterotetramer. The, neuroligin-1/neurexin-1beta complex exhibits a nanomolar affinity and, includes a large binding interface that contains bound Ca(2+)., Alternatively spliced sites in neurexin-1beta and in neuroligin-1 are, positioned nearby the binding interface, explaining how they regulate the, interaction. Structure-based mutations of neuroligin-1 at the interface, disrupt binding to neurexin-1beta, but not the folding of neuroligin-1 and, confirm the validity of the binding interface of the, neuroligin-1/neurexin-1beta complex. Our results provide molecular, insights for understanding the role of cell-adhesion proteins in synapse, function.
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Neurexins and neuroligins provide trans-synaptic connectivity by the Ca2+-dependent interaction of their alternatively spliced extracellular domains. Neuroligins specify synapses in an activity-dependent manner, presumably by binding to neurexins. Here, we present the crystal structures of neuroligin-1 in isolation and in complex with neurexin-1 beta. Neuroligin-1 forms a constitutive dimer, and two neurexin-1 beta monomers bind to two identical surfaces on the opposite faces of the neuroligin-1 dimer to form a heterotetramer. The neuroligin-1/neurexin-1 beta complex exhibits a nanomolar affinity and includes a large binding interface that contains bound Ca2+. Alternatively spliced sites in neurexin-1 beta and in neuroligin-1 are positioned nearby the binding interface, explaining how they regulate the interaction. Structure-based mutations of neuroligin-1 at the interface disrupt binding to neurexin-1 beta, but not the folding of neuroligin-1 and confirm the validity of the binding interface of the neuroligin-1/neurexin-1 beta complex. Our results provide molecular insights for understanding the role of cell-adhesion proteins in synapse function.
==About this Structure==
==About this Structure==
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==Reference==
==Reference==
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Structures of Neuroligin-1 and the Neuroligin-1/Neurexin-1beta Complex Reveal Specific Protein-Protein and Protein-Ca(2+) Interactions., Arac D, Boucard AA, Ozkan E, Strop P, Newell E, Sudhof TC, Brunger AT, Neuron. 2007 Dec 20;56(6):992-1003. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=18093522 18093522]
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Structures of neuroligin-1 and the neuroligin-1/neurexin-1 beta complex reveal specific protein-protein and protein-Ca2+ interactions., Arac D, Boucard AA, Ozkan E, Strop P, Newell E, Sudhof TC, Brunger AT, Neuron. 2007 Dec 20;56(6):992-1003. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=18093522 18093522]
[[Category: Rattus norvegicus]]
[[Category: Rattus norvegicus]]
[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Arac, D.]]
[[Category: Arac, D.]]
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[[Category: Boucard, A.A.]]
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[[Category: Boucard, A A.]]
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[[Category: Brunger, A.T.]]
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[[Category: Brunger, A T.]]
[[Category: Newell, E.]]
[[Category: Newell, E.]]
[[Category: Ozkan, E.]]
[[Category: Ozkan, E.]]
[[Category: Strop, P.]]
[[Category: Strop, P.]]
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[[Category: Sudhof, T.C.]]
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[[Category: Sudhof, T C.]]
[[Category: EDO]]
[[Category: EDO]]
[[Category: NAG]]
[[Category: NAG]]
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[[Category: transmembrane]]
[[Category: transmembrane]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Wed Jan 23 11:14:12 2008''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 19:06:04 2008''

Revision as of 17:06, 21 February 2008


3bix, resolution 1.800Å

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Crystal structure of the extracellular esterase domain of Neuroligin-1

Overview

Neurexins and neuroligins provide trans-synaptic connectivity by the Ca2+-dependent interaction of their alternatively spliced extracellular domains. Neuroligins specify synapses in an activity-dependent manner, presumably by binding to neurexins. Here, we present the crystal structures of neuroligin-1 in isolation and in complex with neurexin-1 beta. Neuroligin-1 forms a constitutive dimer, and two neurexin-1 beta monomers bind to two identical surfaces on the opposite faces of the neuroligin-1 dimer to form a heterotetramer. The neuroligin-1/neurexin-1 beta complex exhibits a nanomolar affinity and includes a large binding interface that contains bound Ca2+. Alternatively spliced sites in neurexin-1 beta and in neuroligin-1 are positioned nearby the binding interface, explaining how they regulate the interaction. Structure-based mutations of neuroligin-1 at the interface disrupt binding to neurexin-1 beta, but not the folding of neuroligin-1 and confirm the validity of the binding interface of the neuroligin-1/neurexin-1 beta complex. Our results provide molecular insights for understanding the role of cell-adhesion proteins in synapse function.

About this Structure

3BIX is a Single protein structure of sequence from Rattus norvegicus with , and as ligands. Full crystallographic information is available from OCA.

Reference

Structures of neuroligin-1 and the neuroligin-1/neurexin-1 beta complex reveal specific protein-protein and protein-Ca2+ interactions., Arac D, Boucard AA, Ozkan E, Strop P, Newell E, Sudhof TC, Brunger AT, Neuron. 2007 Dec 20;56(6):992-1003. PMID:18093522

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