4bxu
From Proteopedia
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| - | '''Unreleased structure''' | ||
| - | + | ==Structure of Pex14 in complex with Pex5 LVxEF motif== | |
| - | + | <StructureSection load='4bxu' size='340' side='right'caption='[[4bxu]]' scene=''> | |
| - | + | == Structural highlights == | |
| - | + | <table><tr><td colspan='2'>[[4bxu]] is a 2 chain structure with sequence from [https://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full experimental information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=4BXU OCA]. For a <b>guided tour on the structure components</b> use [https://proteopedia.org/fgij/fg.htm?mol=4BXU FirstGlance]. <br> | |
| - | + | </td></tr><tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=4bxu FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=4bxu OCA], [https://pdbe.org/4bxu PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=4bxu RCSB], [https://www.ebi.ac.uk/pdbsum/4bxu PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=4bxu ProSAT]</span></td></tr> | |
| + | </table> | ||
| + | == Disease == | ||
| + | [[https://www.uniprot.org/uniprot/PEX14_HUMAN PEX14_HUMAN]] Zellweger syndrome;Neonatal adrenoleukodystrophy;Infantile Refsum disease. The disease is caused by mutations affecting the gene represented in this entry. The disease is caused by mutations affecting the gene represented in this entry. | ||
| + | == Function == | ||
| + | [[https://www.uniprot.org/uniprot/PEX14_HUMAN PEX14_HUMAN]] Component of the peroxisomal translocation machinery with PEX13 and PEX17. Interacts with both the PTS1 and PTS2 receptors. Binds directly to PEX17. | ||
| + | __TOC__ | ||
| + | </StructureSection> | ||
| + | [[Category: Homo sapiens]] | ||
| + | [[Category: Large Structures]] | ||
| + | [[Category: Kooshapur H]] | ||
| + | [[Category: Madl T]] | ||
| + | [[Category: Meyer HN]] | ||
| + | [[Category: Sattler M]] | ||
Current revision
Structure of Pex14 in complex with Pex5 LVxEF motif
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