2h9x

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==Overview==
==Overview==
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CgNa is a toxin from the sea anemone Condylactis gigantea that is, comprised of 47-residues. The structure of CgNa, solved by 1H NMR, spectroscopy, is somewhat atypical, displaying significant homologies to, both type I and II anemonae toxins, as well as a considerable number of, exceptions to what are considered as canonical structural elements of this, group of toxins, and that are thought to be essential for their activity., Furthermore, unique residues in CgNa define a characteristic structure, with strong negatively charged surface-patches. These patches disrupt a, surface-exposed cluster of hydrophobic residues present in all the toxins, described in this group to date. A thorough characterization by, patch-clamp using rat dorsal root ganglion neurons indicate that CgNa, preferentially binds to TTX-S Na + channels in the resting state. This, association increases the inactivation time constant and the rate of, recovery from inactivation, inducing a significant shift to the left in, the steady state of the inactivation curve. The specific structural, features of CgNa may explain its weaker inhibitory capacity when compared, to the other type I and II anemone toxins.
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CgNa (Condylactis gigantea neurotoxin) is a 47-amino-acid- residue toxin from the giant Caribbean sea anemone Condylactis gigantea. The structure of CgNa, which was solved by 1H-NMR spectroscopy, is somewhat atypical and displays significant homology with both type I and II anemone toxins. CgNa also displays a considerable number of exceptions to the canonical structural elements that are thought to be essential for the activity of this group of toxins. Furthermore, unique residues in CgNa define a characteristic structure with strong negatively charged surface patches. These patches disrupt a surface-exposed cluster of hydrophobic residues present in all anemone-derived toxins described to date. A thorough characterization by patch-clamp analysis using rat DRG (dorsal root ganglion) neurons indicated that CgNa preferentially binds to TTX-S (tetrodotoxin-sensitive) voltage-gated sodium channels in the resting state. This association increased the inactivation time constant and the rate of recovery from inactivation, inducing a significant shift in the steady state of inactivation curve to the left. The specific structural features of CgNa may explain its weaker inhibitory capacity when compared with the other type I and II anemone toxins.
==About this Structure==
==About this Structure==
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==Reference==
==Reference==
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CgNa, a type I toxin from the sea anemone Condylactis gigantea shows structural similarities to both type I and II toxins, as well as distinctive structural and functional properties., Salceda E, Perez-Castells J, Lopez-Mendez B, Garateix A, Salazar H, Lopez O, Aneiros A, Standker L, Beress L, Forssmann WG, Soto E, Jimenez-Barbero J, Gimenez-Gallego G, Biochem J. 2007 May 16;. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=17506725 17506725]
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CgNa, a type I toxin from the giant Caribbean sea anemone Condylactis gigantea shows structural similarities to both type I and II toxins, as well as distinctive structural and functional properties(1)., Salceda E, Perez-Castells J, Lopez-Mendez B, Garateix A, Salazar H, Lopez O, Aneiros A, Standker L, Beress L, Forssmann WG, Soto E, Jimenez-Barbero J, Gimenez-Gallego G, Biochem J. 2007 Aug 15;406(1):67-76. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=17506725 17506725]
[[Category: Condylactis gigantea]]
[[Category: Condylactis gigantea]]
[[Category: Single protein]]
[[Category: Single protein]]
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[[Category: beta sheet]]
[[Category: beta sheet]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Wed Jan 23 15:00:19 2008''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 17:39:43 2008''

Revision as of 15:39, 21 February 2008


2h9x

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NMR structure for the CgNa toxin from the sea anemone Condylactis gigantea

Overview

CgNa (Condylactis gigantea neurotoxin) is a 47-amino-acid- residue toxin from the giant Caribbean sea anemone Condylactis gigantea. The structure of CgNa, which was solved by 1H-NMR spectroscopy, is somewhat atypical and displays significant homology with both type I and II anemone toxins. CgNa also displays a considerable number of exceptions to the canonical structural elements that are thought to be essential for the activity of this group of toxins. Furthermore, unique residues in CgNa define a characteristic structure with strong negatively charged surface patches. These patches disrupt a surface-exposed cluster of hydrophobic residues present in all anemone-derived toxins described to date. A thorough characterization by patch-clamp analysis using rat DRG (dorsal root ganglion) neurons indicated that CgNa preferentially binds to TTX-S (tetrodotoxin-sensitive) voltage-gated sodium channels in the resting state. This association increased the inactivation time constant and the rate of recovery from inactivation, inducing a significant shift in the steady state of inactivation curve to the left. The specific structural features of CgNa may explain its weaker inhibitory capacity when compared with the other type I and II anemone toxins.

About this Structure

2H9X is a Single protein structure of sequence from Condylactis gigantea. Full crystallographic information is available from OCA.

Reference

CgNa, a type I toxin from the giant Caribbean sea anemone Condylactis gigantea shows structural similarities to both type I and II toxins, as well as distinctive structural and functional properties(1)., Salceda E, Perez-Castells J, Lopez-Mendez B, Garateix A, Salazar H, Lopez O, Aneiros A, Standker L, Beress L, Forssmann WG, Soto E, Jimenez-Barbero J, Gimenez-Gallego G, Biochem J. 2007 Aug 15;406(1):67-76. PMID:17506725

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