Outer surface protein

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[[1osp]], [[1fj1]] – OspA (mutant) + antibody – ''Borrelia burgdorferi''<br />
[[1osp]], [[1fj1]] – OspA (mutant) + antibody – ''Borrelia burgdorferi''<br />
-
[[2af5]], [[2fkg]], [[2fkj]], [[2hkd]] – OspA<br />
+
[[2af5]], [[2fkg]], [[2fkj]], [[2hkd]], [[2oyb]] – OspA<br />
-
[[2g8c]], [[2i5z]], [[2ol6]], [[2ol7]], [[2ol8]], [[2oy1]], [[2i5v]], [[2oy5]], [[2oy7]], [[2oy8]] - OspA (mutant) <br />
+
[[2g8c]], [[2i5z]], [[2ol6]], [[2ol7]], [[2ol8]], [[2oy1]], [[2i5v]], [[2oy5]], [[2oy7]], [[2oy8]], [[2pi3]], [[3aum]], [[3cka]], [[3ckf]], [[3ckg]], [[3ec5]], [[3eex]] - OspA (mutant) <br />
===OspB===
===OspB===
[[1p4p]] – OspB C terminal<br />
[[1p4p]] – OspB C terminal<br />
-
[[1rjl]] - OspB+ antibody
+
[[1rjl]] - OspB+ antibody<br />
 +
[[2yn7]] – OspB<br />
===OspC===
===OspC===
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[[1ggq]], [[1g5z]] – OspC<br />
[[1ggq]], [[1g5z]] – OspC<br />
[[1f1m]] – OspC (mutant)
[[1f1m]] – OspC (mutant)
 +
 +
===OspE===
 +
 +
[[2m4f]] – OspE – NMR<br />
 +
[[4j38]] – OspE + complement factor H<br />
 +
[[Category:Topic Page]]
[[Category:Topic Page]]

Revision as of 09:31, 1 April 2014

Template:STRUCTURE 2af5


Outer surface proteins (Osp) are produced by the bacteria Borrelia burgdorferi which causes Lyme disease. OspA and OspB are upregulated when the bacteria enter the tick. OspA contributes to the colonization of the gut by the bacteria. OspC is a major antigen on the surface of the Lyme disease spirochetes when it is being transmitted to humans. For more details see:
OspA -

OspB -

OspC -

OxpA, OspB and OspC -


Contents

3D structures of outer surface protein

Updated on 01-April-2014

OspA

1osp, 1fj1 – OspA (mutant) + antibody – Borrelia burgdorferi
2af5, 2fkg, 2fkj, 2hkd, 2oyb – OspA
2g8c, 2i5z, 2ol6, 2ol7, 2ol8, 2oy1, 2i5v, 2oy5, 2oy7, 2oy8, 2pi3, 3aum, 3cka, 3ckf, 3ckg, 3ec5, 3eex - OspA (mutant)

OspB

1p4p – OspB C terminal
1rjl - OspB+ antibody
2yn7 – OspB

OspC

1ggq, 1g5z – OspC
1f1m – OspC (mutant)

OspE

2m4f – OspE – NMR
4j38 – OspE + complement factor H

Proteopedia Page Contributors and Editors (what is this?)

Michal Harel, Joel L. Sussman, Alexander Berchansky

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