4unl

<table><tr><td colspan='2'>[[4unl]] is a 2 chain structure with sequence from [http://en.wikipedia.org/wiki/Human Human]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=4UNL OCA]. For a <b>guided tour on the structure components</b> use [http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=4UNL FirstGlance]. <br>

</td></tr><tr id='related'><td class="sblockLbl"><b>[[Related_structure|Related:]]</b></td><td class="sblockDat">[[4unk|4unk]]</td></tr>

<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=4unl FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=4unl OCA], [http://pdbe.org/4unl PDBe], [http://www.rcsb.org/pdb/explore.do?structureId=4unl RCSB], [http://www.ebi.ac.uk/pdbsum/4unl PDBsum], [http://prosat.h-its.org/prosat/prosatexe?pdbcode=4unl ProSAT]</span></td></tr>

[[http://www.uniprot.org/uniprot/TPIS_HUMAN TPIS_HUMAN]] Defects in TPI1 are the cause of triosephosphate isomerase deficiency (TPI deficiency) [MIM:[http://omim.org/entry/190450 190450]]. TPI deficiency is an autosomal recessive disorder. It is the most severe clinical disorder of glycolysis. It is associated with neonatal jaundice, chronic hemolytic anemia, progressive neuromuscular dysfunction, cardiomyopathy and increased susceptibility to infection.

*[[Triose Phosphate Isomerase|Triose Phosphate Isomerase]]

[[Category: Human]]

[[Category: Castillo-Villanueva, A]]

[[Category: DeLaMora-DeLaMora, I]]

[[Category: Enriquez-Flores, S]]

[[Category: Garcia-Torres, I]]

[[Category: Gomez-Manzo, S]]

[[Category: Lopez-Velazquez, G]]

[[Category: Marcial-Quino, J]]

[[Category: Mendez, S T]]

[[Category: Oria-Hernandez, J]]

[[Category: Reyes-Vivas, H]]

[[Category: Torres-Arroyo, A]]

[[Category: Torres-Larios, A]]

[[Category: Isomerase]]