Hemagglutinin-esterase
From Proteopedia
(Difference between revisions)
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</StructureSection> | </StructureSection> | ||
| - | ==3D structures of hemagglutinin-esterase== | ||
| - | Updated on {{REVISIONDAY2}}-{{MONTHNAME|{{REVISIONMONTH}}}}-{{REVISIONYEAR}} | ||
| - | {{#tree:id=OrganizedByTopic|openlevels=0| | ||
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| - | *Hemagglutinin-esterase | ||
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| - | **[[3cl4]] – bcvHE – bovine corona virus<br /> | ||
| - | **[[3i1k]] – PtHE (mutant) – porcine torovirus<br /> | ||
| - | **[[3i26]] – BtHE – bovine torovirus<br /> | ||
| - | **[[4c7l]] – mhvHE (mutant) – murine hepatitis virus<br /> | ||
| - | **[[5t9y]], [[5t96]] – savHE – salmon anemia virus<br /> | ||
| - | **[[5n11]] – HE – human corona virus<br /> | ||
| - | **[[5jif]] – HE – murine corona virus<br /> | ||
| - | **[[4zxn]] – rcvHE (mutant) – rat coronavirus<br /> | ||
| - | **[[5e64]] – IdvHE – Influenza D virus<br /> | ||
| - | **[[5e5w]] – IdvHE (mutant) <br /> | ||
| - | |||
| - | *Hemagglutinin-esterase complex | ||
| - | |||
| - | **[[3cl5]] – bcvHE (mutant) + receptor<br /> | ||
| - | **[[3i1l]] – PtHE (mutant) + receptor<br /> | ||
| - | **[[3i27]] – BtHE + receptor<br /> | ||
| - | **[[4c7w]] – mhvHE (mutant) + receptor<br /> | ||
| - | **[[5t9y]] – savHE – salmon anemia virus<br /> | ||
| - | **[[5jil]] – rcvHE (mutant) + sialic acid derivative <br /> | ||
| - | **[[5e62]] – IdvHE + glucopyranoside derivative<br /> | ||
| - | **[[5e66]] – IdvHE + receptor analog<br /> | ||
| - | **[[5e65]] – IdvHE (mutant) + receptor analog<br /> | ||
| - | }} | ||
== References == | == References == | ||
<references/> | <references/> | ||
[[Category:Topic Page]] | [[Category:Topic Page]] | ||
Current revision
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References
- ↑ de Groot RJ. Structure, function and evolution of the hemagglutinin-esterase proteins of corona- and toroviruses. Glycoconj J. 2006 Feb;23(1-2):59-72. PMID:16575523 doi:http://dx.doi.org/10.1007/s10719-006-5438-8
- ↑ Langereis MA, Zeng Q, Heesters BA, Huizinga EG, de Groot RJ. The murine coronavirus hemagglutinin-esterase receptor-binding site: a major shift in ligand specificity through modest changes in architecture. PLoS Pathog. 2012 Jan;8(1):e1002492. doi: 10.1371/journal.ppat.1002492. Epub 2012, Jan 26. PMID:22291594 doi:http://dx.doi.org/10.1371/journal.ppat.1002492
