1i4m

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(New page: 200px<br /> <applet load="1i4m" size="450" color="white" frame="true" align="right" spinBox="true" caption="1i4m, resolution 2.00&Aring;" /> '''Crystal structure o...)
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[[Image:1i4m.gif|left|200px]]<br />
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[[Image:1i4m.gif|left|200px]]<br /><applet load="1i4m" size="350" color="white" frame="true" align="right" spinBox="true"
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<applet load="1i4m" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="1i4m, resolution 2.00&Aring;" />
caption="1i4m, resolution 2.00&Aring;" />
'''Crystal structure of the human prion protein reveals a mechanism for oligomerization'''<br />
'''Crystal structure of the human prion protein reveals a mechanism for oligomerization'''<br />
==Overview==
==Overview==
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The pathogenesis of transmissible encephalopathies is associated with the, conversion of the cellular prion protein, PrP(C), into a conformationally, altered oligomeric form, PrP(Sc). Here we report the crystal structure of, the human prion protein in dimer form at 2 A resolution. The dimer results, from the three-dimensional swapping of the C-terminal helix 3 and, rearrangement of the disulfide bond. An interchain two-stranded, antiparallel beta-sheet is formed at the dimer interface by residues that, are located in helix 2 in the monomeric NMR structures. Familial prion, disease mutations map to the regions directly involved in helix swapping., This crystal structure suggests that oligomerization through 3D, domain-swapping may constitute an important step on the pathway of the, PrP(C) --&gt; PrP(Sc) conversion.
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The pathogenesis of transmissible encephalopathies is associated with the conversion of the cellular prion protein, PrP(C), into a conformationally altered oligomeric form, PrP(Sc). Here we report the crystal structure of the human prion protein in dimer form at 2 A resolution. The dimer results from the three-dimensional swapping of the C-terminal helix 3 and rearrangement of the disulfide bond. An interchain two-stranded antiparallel beta-sheet is formed at the dimer interface by residues that are located in helix 2 in the monomeric NMR structures. Familial prion disease mutations map to the regions directly involved in helix swapping. This crystal structure suggests that oligomerization through 3D domain-swapping may constitute an important step on the pathway of the PrP(C) --&gt; PrP(Sc) conversion.
==Disease==
==Disease==
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==About this Structure==
==About this Structure==
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1I4M is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with CD and CL as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1I4M OCA].
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1I4M is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=CD:'>CD</scene> and <scene name='pdbligand=CL:'>CL</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1I4M OCA].
==Reference==
==Reference==
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[[Category: Homo sapiens]]
[[Category: Homo sapiens]]
[[Category: Single protein]]
[[Category: Single protein]]
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[[Category: Knaus, K.J.]]
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[[Category: Knaus, K J.]]
[[Category: Malone, M.]]
[[Category: Malone, M.]]
[[Category: Morillas, M.]]
[[Category: Morillas, M.]]
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[[Category: Surewicz, W.K.]]
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[[Category: Surewicz, W K.]]
[[Category: Swietnicki, W.]]
[[Category: Swietnicki, W.]]
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[[Category: Yee, V.C.]]
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[[Category: Yee, V C.]]
[[Category: CD]]
[[Category: CD]]
[[Category: CL]]
[[Category: CL]]
[[Category: domain-swapped dimer]]
[[Category: domain-swapped dimer]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 17:26:17 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 13:07:58 2008''

Revision as of 11:07, 21 February 2008


1i4m, resolution 2.00Å

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Crystal structure of the human prion protein reveals a mechanism for oligomerization

Contents

Overview

The pathogenesis of transmissible encephalopathies is associated with the conversion of the cellular prion protein, PrP(C), into a conformationally altered oligomeric form, PrP(Sc). Here we report the crystal structure of the human prion protein in dimer form at 2 A resolution. The dimer results from the three-dimensional swapping of the C-terminal helix 3 and rearrangement of the disulfide bond. An interchain two-stranded antiparallel beta-sheet is formed at the dimer interface by residues that are located in helix 2 in the monomeric NMR structures. Familial prion disease mutations map to the regions directly involved in helix swapping. This crystal structure suggests that oligomerization through 3D domain-swapping may constitute an important step on the pathway of the PrP(C) --> PrP(Sc) conversion.

Disease

Known diseases associated with this structure: Creutzfeldt-Jakob disease OMIM:[176640], Gerstmann-Straussler disease OMIM:[176640], Huntington disease-like 1 OMIM:[176640], Insomnia, fatal familial OMIM:[176640], Prion disease with protracted course OMIM:[176640], Retinitis pigmentosa-11 OMIM:[606419]

About this Structure

1I4M is a Single protein structure of sequence from Homo sapiens with and as ligands. Full crystallographic information is available from OCA.

Reference

Crystal structure of the human prion protein reveals a mechanism for oligomerization., Knaus KJ, Morillas M, Swietnicki W, Malone M, Surewicz WK, Yee VC, Nat Struct Biol. 2001 Sep;8(9):770-4. PMID:11524679

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