2a1u

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(New page: 200px<br /> <applet load="2a1u" size="450" color="white" frame="true" align="right" spinBox="true" caption="2a1u, resolution 2.11&Aring;" /> '''Crystal structure o...)
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[[Image:2a1u.gif|left|200px]]<br />
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[[Image:2a1u.gif|left|200px]]<br /><applet load="2a1u" size="350" color="white" frame="true" align="right" spinBox="true"
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<applet load="2a1u" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="2a1u, resolution 2.11&Aring;" />
caption="2a1u, resolution 2.11&Aring;" />
'''Crystal structure of the human ETF E165betaA mutant'''<br />
'''Crystal structure of the human ETF E165betaA mutant'''<br />
==Overview==
==Overview==
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Crystal structures of protein complexes with electron-transferring, flavoprotein (ETF) have revealed a dual protein-protein interface with one, region serving as anchor while the ETF FAD domain samples available space, within the complex. We show that mutation of the conserved Glu-165beta in, human ETF leads to drastically modulated rates of interprotein electron, transfer with both medium chain acyl-CoA dehydrogenase and dimethylglycine, dehydrogenase. The crystal structure of free E165betaA ETF is essentially, identical to that of wild-type ETF, but the crystal structure of the, E165betaA ETF.medium chain acyl-CoA dehydrogenase complex reveals clear, electron density for the FAD domain in a position optimal for fast, interprotein electron transfer. Based on our observations, we present a, dynamic multistate model for conformational sampling that for the, wild-type ETF. medium chain acyl-CoA dehydrogenase complex involves random, motion between three distinct positions for the ETF FAD domain. ETF, Glu-165beta plays a key role in stabilizing positions incompatible with, fast interprotein electron transfer, thus ensuring high rates of complex, dissociation.
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Crystal structures of protein complexes with electron-transferring flavoprotein (ETF) have revealed a dual protein-protein interface with one region serving as anchor while the ETF FAD domain samples available space within the complex. We show that mutation of the conserved Glu-165beta in human ETF leads to drastically modulated rates of interprotein electron transfer with both medium chain acyl-CoA dehydrogenase and dimethylglycine dehydrogenase. The crystal structure of free E165betaA ETF is essentially identical to that of wild-type ETF, but the crystal structure of the E165betaA ETF.medium chain acyl-CoA dehydrogenase complex reveals clear electron density for the FAD domain in a position optimal for fast interprotein electron transfer. Based on our observations, we present a dynamic multistate model for conformational sampling that for the wild-type ETF. medium chain acyl-CoA dehydrogenase complex involves random motion between three distinct positions for the ETF FAD domain. ETF Glu-165beta plays a key role in stabilizing positions incompatible with fast interprotein electron transfer, thus ensuring high rates of complex dissociation.
==Disease==
==Disease==
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==About this Structure==
==About this Structure==
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2A1U is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with FAD and AMP as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=2A1U OCA].
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2A1U is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=FAD:'>FAD</scene> and <scene name='pdbligand=AMP:'>AMP</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2A1U OCA].
==Reference==
==Reference==
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[[Category: Protein complex]]
[[Category: Protein complex]]
[[Category: Leys, D.]]
[[Category: Leys, D.]]
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[[Category: Scrutton, N.S.]]
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[[Category: Scrutton, N S.]]
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[[Category: Thiel, A.Van.]]
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[[Category: Thiel, A Van.]]
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[[Category: Toogood, H.S.]]
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[[Category: Toogood, H S.]]
[[Category: AMP]]
[[Category: AMP]]
[[Category: FAD]]
[[Category: FAD]]
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[[Category: mobile domain]]
[[Category: mobile domain]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 20:44:48 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 16:23:01 2008''

Revision as of 14:23, 21 February 2008


2a1u, resolution 2.11Å

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Crystal structure of the human ETF E165betaA mutant

Contents

Overview

Crystal structures of protein complexes with electron-transferring flavoprotein (ETF) have revealed a dual protein-protein interface with one region serving as anchor while the ETF FAD domain samples available space within the complex. We show that mutation of the conserved Glu-165beta in human ETF leads to drastically modulated rates of interprotein electron transfer with both medium chain acyl-CoA dehydrogenase and dimethylglycine dehydrogenase. The crystal structure of free E165betaA ETF is essentially identical to that of wild-type ETF, but the crystal structure of the E165betaA ETF.medium chain acyl-CoA dehydrogenase complex reveals clear electron density for the FAD domain in a position optimal for fast interprotein electron transfer. Based on our observations, we present a dynamic multistate model for conformational sampling that for the wild-type ETF. medium chain acyl-CoA dehydrogenase complex involves random motion between three distinct positions for the ETF FAD domain. ETF Glu-165beta plays a key role in stabilizing positions incompatible with fast interprotein electron transfer, thus ensuring high rates of complex dissociation.

Disease

Known diseases associated with this structure: Glutaricaciduria, type IIA OMIM:[608053], Glutaricaciduria, type IIB OMIM:[130410]

About this Structure

2A1U is a Protein complex structure of sequences from Homo sapiens with and as ligands. Full crystallographic information is available from OCA.

Reference

Stabilization of non-productive conformations underpins rapid electron transfer to electron-transferring flavoprotein., Toogood HS, van Thiel A, Scrutton NS, Leys D, J Biol Chem. 2005 Aug 26;280(34):30361-6. Epub 2005 Jun 23. PMID:15975918

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