2i83

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(New page: 200px<br /> <applet load="2i83" size="450" color="white" frame="true" align="right" spinBox="true" caption="2i83" /> '''hyaluronan-binding domain of CD44 in its li...)
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'''hyaluronan-binding domain of CD44 in its ligand-bound form'''<br />
'''hyaluronan-binding domain of CD44 in its ligand-bound form'''<br />
==Overview==
==Overview==
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CD44, a major cell surface receptor for hyaluronan (HA), contains a, functional domain responsible for HA binding at its N terminus (residues, 21-178). Accumulating evidence indicates that proteolytic cleavage of CD44, in its extracellular region (residues 21-268) leads to enhanced tumor cell, migration and invasion. Hence, understanding the mechanisms underlying the, CD44 proteolytic cleavage is important for understanding the mechanism of, CD44-mediated tumor progression. Here we present the NMR structure of the, HA-binding domain of CD44 in its HA-bound state. The structure is composed, of the Link module (residues 32-124) and an extended lobe (residues 21-31, and 125-152). Interestingly, a comparison of its unbound and HA-bound, structures revealed that rearrangement of the beta-strands in the extended, lobe (residues 143-148) and disorder of the structure in the following, C-terminal region (residues 153-169) occurred upon HA binding, which is, consistent with the results of trypsin proteolysis studies of the CD44, HA-binding domain. The order-to-disorder transition of the C-terminal, region by HA binding may be involved in the CD44-mediated cell migration.
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CD44, a major cell surface receptor for hyaluronan (HA), contains a functional domain responsible for HA binding at its N terminus (residues 21-178). Accumulating evidence indicates that proteolytic cleavage of CD44 in its extracellular region (residues 21-268) leads to enhanced tumor cell migration and invasion. Hence, understanding the mechanisms underlying the CD44 proteolytic cleavage is important for understanding the mechanism of CD44-mediated tumor progression. Here we present the NMR structure of the HA-binding domain of CD44 in its HA-bound state. The structure is composed of the Link module (residues 32-124) and an extended lobe (residues 21-31 and 125-152). Interestingly, a comparison of its unbound and HA-bound structures revealed that rearrangement of the beta-strands in the extended lobe (residues 143-148) and disorder of the structure in the following C-terminal region (residues 153-169) occurred upon HA binding, which is consistent with the results of trypsin proteolysis studies of the CD44 HA-binding domain. The order-to-disorder transition of the C-terminal region by HA binding may be involved in the CD44-mediated cell migration.
==Disease==
==Disease==
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==About this Structure==
==About this Structure==
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2I83 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=2I83 OCA].
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2I83 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2I83 OCA].
==Reference==
==Reference==
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[[Category: Sakakura, M.]]
[[Category: Sakakura, M.]]
[[Category: Shimada, I.]]
[[Category: Shimada, I.]]
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[[Category: Sugahara, K.N.]]
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[[Category: Sugahara, K N.]]
[[Category: Takeda, M.]]
[[Category: Takeda, M.]]
[[Category: Terasawa, H.]]
[[Category: Terasawa, H.]]
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[[Category: link module]]
[[Category: link module]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 22:42:22 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 17:50:06 2008''

Revision as of 15:50, 21 February 2008


2i83

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hyaluronan-binding domain of CD44 in its ligand-bound form

Contents

Overview

CD44, a major cell surface receptor for hyaluronan (HA), contains a functional domain responsible for HA binding at its N terminus (residues 21-178). Accumulating evidence indicates that proteolytic cleavage of CD44 in its extracellular region (residues 21-268) leads to enhanced tumor cell migration and invasion. Hence, understanding the mechanisms underlying the CD44 proteolytic cleavage is important for understanding the mechanism of CD44-mediated tumor progression. Here we present the NMR structure of the HA-binding domain of CD44 in its HA-bound state. The structure is composed of the Link module (residues 32-124) and an extended lobe (residues 21-31 and 125-152). Interestingly, a comparison of its unbound and HA-bound structures revealed that rearrangement of the beta-strands in the extended lobe (residues 143-148) and disorder of the structure in the following C-terminal region (residues 153-169) occurred upon HA binding, which is consistent with the results of trypsin proteolysis studies of the CD44 HA-binding domain. The order-to-disorder transition of the C-terminal region by HA binding may be involved in the CD44-mediated cell migration.

Disease

Known diseases associated with this structure: Blood group, Indian system OMIM:[107269], Fertile eunuch syndrome OMIM:[138850], Hypogonadotropic hypogonadism OMIM:[138850]

About this Structure

2I83 is a Single protein structure of sequence from Homo sapiens. Full crystallographic information is available from OCA.

Reference

Ligand-induced structural changes of the CD44 hyaluronan-binding domain revealed by NMR., Takeda M, Ogino S, Umemoto R, Sakakura M, Kajiwara M, Sugahara KN, Hayasaka H, Miyasaka M, Terasawa H, Shimada I, J Biol Chem. 2006 Dec 29;281(52):40089-95. Epub 2006 Nov 2. PMID:17085435

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