7nn9

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(New page: 200px<br /><applet load="7nn9" size="450" color="white" frame="true" align="right" spinBox="true" caption="7nn9, resolution 2.&Aring;" /> '''NATIVE INFLUENZA VIRUS...)
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'''NATIVE INFLUENZA VIRUS NEURAMINIDASE SUBTYPE N9 (TERN)'''<br />
'''NATIVE INFLUENZA VIRUS NEURAMINIDASE SUBTYPE N9 (TERN)'''<br />
==Overview==
==Overview==
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The three-dimensional X-ray structure of a complex of the potent, neuraminidase inhibitor 4-guanidino-Neu5Ac2en and influenza virus, neuraminidase (Subtype N9) has been obtained utilizing diffraction data to, 1.8 A resolution. The interactions of the inhibitor, solvent water, molecules, and the active site residues have been accurately determined., Six water molecules bound in the native structure have been displaced by, the inhibitor, and the active site residues show no significant, conformational changes on binding. Sialic acid, the natural substrate, binds in a half-chair conformation that is isosteric to the inhibitor. The, conformation of the inhibitor in the active site of the X-ray structure, concurs with that obtained by theoretical calculations and validates the, structure-based design of the inhibitor. Comparison of known, high-resolution structures of neuraminidase subtypes N2, N9, and B shows, good structural conservation of the active site protein atoms, but the, location of the water molecules in the respective active sites is less, conserved. In particular, the environment of the 4-guanidino group of the, inhibitor is strongly conserved and is the basis for the antiviral action, of the inhibitor across all presently known influenza strains. Differences, in the solvent structure in the active site may be related to variation in, the affinities of inhibitors to different subtypes of neuraminidase.
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The three-dimensional X-ray structure of a complex of the potent neuraminidase inhibitor 4-guanidino-Neu5Ac2en and influenza virus neuraminidase (Subtype N9) has been obtained utilizing diffraction data to 1.8 A resolution. The interactions of the inhibitor, solvent water molecules, and the active site residues have been accurately determined. Six water molecules bound in the native structure have been displaced by the inhibitor, and the active site residues show no significant conformational changes on binding. Sialic acid, the natural substrate, binds in a half-chair conformation that is isosteric to the inhibitor. The conformation of the inhibitor in the active site of the X-ray structure concurs with that obtained by theoretical calculations and validates the structure-based design of the inhibitor. Comparison of known high-resolution structures of neuraminidase subtypes N2, N9, and B shows good structural conservation of the active site protein atoms, but the location of the water molecules in the respective active sites is less conserved. In particular, the environment of the 4-guanidino group of the inhibitor is strongly conserved and is the basis for the antiviral action of the inhibitor across all presently known influenza strains. Differences in the solvent structure in the active site may be related to variation in the affinities of inhibitors to different subtypes of neuraminidase.
==About this Structure==
==About this Structure==
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7NN9 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Unidentified_influenza_virus Unidentified influenza virus] with NAG and CA as [http://en.wikipedia.org/wiki/ligands ligands]. This structure superseeds the now removed PDB entry 2NN9. Active as [http://en.wikipedia.org/wiki/Exo-alpha-sialidase Exo-alpha-sialidase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.2.1.18 3.2.1.18] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=7NN9 OCA].
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7NN9 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Unidentified_influenza_virus Unidentified influenza virus] with <scene name='pdbligand=NAG:'>NAG</scene> and <scene name='pdbligand=CA:'>CA</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. This structure supersedes the now removed PDB entry 2NN9. Active as [http://en.wikipedia.org/wiki/Exo-alpha-sialidase Exo-alpha-sialidase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.2.1.18 3.2.1.18] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=7NN9 OCA].
==Reference==
==Reference==
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[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Unidentified influenza virus]]
[[Category: Unidentified influenza virus]]
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[[Category: Colman, P.M.]]
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[[Category: Colman, P M.]]
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[[Category: Varghese, J.N.]]
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[[Category: Varghese, J N.]]
[[Category: CA]]
[[Category: CA]]
[[Category: NAG]]
[[Category: NAG]]
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[[Category: sialidase]]
[[Category: sialidase]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Tue Nov 20 15:13:09 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 19:17:28 2008''

Revision as of 17:17, 21 February 2008


7nn9, resolution 2.Å

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NATIVE INFLUENZA VIRUS NEURAMINIDASE SUBTYPE N9 (TERN)

Overview

The three-dimensional X-ray structure of a complex of the potent neuraminidase inhibitor 4-guanidino-Neu5Ac2en and influenza virus neuraminidase (Subtype N9) has been obtained utilizing diffraction data to 1.8 A resolution. The interactions of the inhibitor, solvent water molecules, and the active site residues have been accurately determined. Six water molecules bound in the native structure have been displaced by the inhibitor, and the active site residues show no significant conformational changes on binding. Sialic acid, the natural substrate, binds in a half-chair conformation that is isosteric to the inhibitor. The conformation of the inhibitor in the active site of the X-ray structure concurs with that obtained by theoretical calculations and validates the structure-based design of the inhibitor. Comparison of known high-resolution structures of neuraminidase subtypes N2, N9, and B shows good structural conservation of the active site protein atoms, but the location of the water molecules in the respective active sites is less conserved. In particular, the environment of the 4-guanidino group of the inhibitor is strongly conserved and is the basis for the antiviral action of the inhibitor across all presently known influenza strains. Differences in the solvent structure in the active site may be related to variation in the affinities of inhibitors to different subtypes of neuraminidase.

About this Structure

7NN9 is a Single protein structure of sequence from Unidentified influenza virus with and as ligands. This structure supersedes the now removed PDB entry 2NN9. Active as Exo-alpha-sialidase, with EC number 3.2.1.18 Full crystallographic information is available from OCA.

Reference

Three-dimensional structure of the complex of 4-guanidino-Neu5Ac2en and influenza virus neuraminidase., Varghese JN, Epa VC, Colman PM, Protein Sci. 1995 Jun;4(6):1081-7. PMID:7549872

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