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1oql

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(New page: 200px<br /><applet load="1oql" size="450" color="white" frame="true" align="right" spinBox="true" caption="1oql, resolution 3.0&Aring;" /> '''Mistletoe Lectin I fr...)
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'''Mistletoe Lectin I from Viscum album complexed with galactose'''<br />
'''Mistletoe Lectin I from Viscum album complexed with galactose'''<br />
==Overview==
==Overview==
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The X-ray structure of mistletoe lectin I (MLI), a type-II, ribosome-inactivating protein (RIP), cocrystallized with galactose is, described. The model was refined at 3.0 A resolution to an R-factor of, 19.9% using 21 899 reflections, with Rfree 24.0%. MLI forms a homodimer, (A-B)2 in the crystal, as it does in solution at high concentration. The, dimer is formed through contacts between the N-terminal domains of two, B-chains involving weak polar and non-polar interactions. Consequently, the overall arrangement of sugar-binding sites in MLI differs from those, in monomeric type-II RIPs: two N-terminal sugar-binding sites are 15 A, apart on one side of the dimer, and two C-terminal sugar-binding sites are, 87 A apart on the other side. Galactose binding is achieved by common, hydrogen bonds for the two binding sites via hydroxy groups 3-OH and 4-OH, and hydrophobic contact by an aromatic ring. In addition, at the, N-terminal site 2-OH forms hydrogen bonds with Asp27 and Lys41, and at the, C-terminal site 3-OH and 6-OH undergo water-mediated interactions and C5, has a hydrophobic contact. MLI is a galactose-specific lectin and shows, little affinity for N-acetylgalactosamine. The reason for this is, discussed. Structural differences among the RIPs investigated in this, study (their quaternary structures, location of sugar-binding sites, and, fine sugar specificities of their B-chains, which could have diverged, through evolution from a two-domain protein) may affect the binding sites, and consequently the cellular transport processes and biological responses, of these toxins.
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The X-ray structure of mistletoe lectin I (MLI), a type-II ribosome-inactivating protein (RIP), cocrystallized with galactose is described. The model was refined at 3.0 A resolution to an R-factor of 19.9% using 21 899 reflections, with Rfree 24.0%. MLI forms a homodimer (A-B)2 in the crystal, as it does in solution at high concentration. The dimer is formed through contacts between the N-terminal domains of two B-chains involving weak polar and non-polar interactions. Consequently, the overall arrangement of sugar-binding sites in MLI differs from those in monomeric type-II RIPs: two N-terminal sugar-binding sites are 15 A apart on one side of the dimer, and two C-terminal sugar-binding sites are 87 A apart on the other side. Galactose binding is achieved by common hydrogen bonds for the two binding sites via hydroxy groups 3-OH and 4-OH and hydrophobic contact by an aromatic ring. In addition, at the N-terminal site 2-OH forms hydrogen bonds with Asp27 and Lys41, and at the C-terminal site 3-OH and 6-OH undergo water-mediated interactions and C5 has a hydrophobic contact. MLI is a galactose-specific lectin and shows little affinity for N-acetylgalactosamine. The reason for this is discussed. Structural differences among the RIPs investigated in this study (their quaternary structures, location of sugar-binding sites, and fine sugar specificities of their B-chains, which could have diverged through evolution from a two-domain protein) may affect the binding sites, and consequently the cellular transport processes and biological responses of these toxins.
==About this Structure==
==About this Structure==
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1OQL is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Viscum_album Viscum album] with GAL and NAG as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/rRNA_N-glycosylase rRNA N-glycosylase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.2.2.22 3.2.2.22] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1OQL OCA].
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1OQL is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Viscum_album Viscum album] with <scene name='pdbligand=GAL:'>GAL</scene> and <scene name='pdbligand=NAG:'>NAG</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/rRNA_N-glycosylase rRNA N-glycosylase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.2.2.22 3.2.2.22] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1OQL OCA].
==Reference==
==Reference==
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[[Category: Viscum album]]
[[Category: Viscum album]]
[[Category: rRNA N-glycosylase]]
[[Category: rRNA N-glycosylase]]
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[[Category: Agapov, I.I.]]
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[[Category: Agapov, I I.]]
[[Category: Niwa, H.]]
[[Category: Niwa, H.]]
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[[Category: Palmer, R.A.]]
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[[Category: Palmer, R A.]]
[[Category: Pfuller, U.]]
[[Category: Pfuller, U.]]
[[Category: Saward, S.]]
[[Category: Saward, S.]]
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[[Category: Tonevitsky, A.G.]]
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[[Category: Tonevitsky, A G.]]
[[Category: GAL]]
[[Category: GAL]]
[[Category: NAG]]
[[Category: NAG]]
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[[Category: type-ii ribosome-inactivating protein]]
[[Category: type-ii ribosome-inactivating protein]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Tue Nov 20 23:06:27 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 14:20:31 2008''

Revision as of 12:20, 21 February 2008


1oql, resolution 3.0Å

Drag the structure with the mouse to rotate

Mistletoe Lectin I from Viscum album complexed with galactose

Overview

The X-ray structure of mistletoe lectin I (MLI), a type-II ribosome-inactivating protein (RIP), cocrystallized with galactose is described. The model was refined at 3.0 A resolution to an R-factor of 19.9% using 21 899 reflections, with Rfree 24.0%. MLI forms a homodimer (A-B)2 in the crystal, as it does in solution at high concentration. The dimer is formed through contacts between the N-terminal domains of two B-chains involving weak polar and non-polar interactions. Consequently, the overall arrangement of sugar-binding sites in MLI differs from those in monomeric type-II RIPs: two N-terminal sugar-binding sites are 15 A apart on one side of the dimer, and two C-terminal sugar-binding sites are 87 A apart on the other side. Galactose binding is achieved by common hydrogen bonds for the two binding sites via hydroxy groups 3-OH and 4-OH and hydrophobic contact by an aromatic ring. In addition, at the N-terminal site 2-OH forms hydrogen bonds with Asp27 and Lys41, and at the C-terminal site 3-OH and 6-OH undergo water-mediated interactions and C5 has a hydrophobic contact. MLI is a galactose-specific lectin and shows little affinity for N-acetylgalactosamine. The reason for this is discussed. Structural differences among the RIPs investigated in this study (their quaternary structures, location of sugar-binding sites, and fine sugar specificities of their B-chains, which could have diverged through evolution from a two-domain protein) may affect the binding sites, and consequently the cellular transport processes and biological responses of these toxins.

About this Structure

1OQL is a Protein complex structure of sequences from Viscum album with and as ligands. Active as rRNA N-glycosylase, with EC number 3.2.2.22 Full crystallographic information is available from OCA.

Reference

Crystal structure at 3 A of mistletoe lectin I, a dimeric type-II ribosome-inactivating protein, complexed with galactose., Niwa H, Tonevitsky AG, Agapov II, Saward S, Pfuller U, Palmer RA, Eur J Biochem. 2003 Jul;270(13):2739-49. PMID:12823544

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