3f66
From Proteopedia
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==About this Structure== | ==About this Structure== | ||
| - | 3F66 is a | + | 3F66 is a 2 chains structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=3F66 OCA]. |
==Reference== | ==Reference== | ||
| - | + | <ref group="xtra">PMID:19059779</ref><references group="xtra"/> | |
[[Category: Homo sapiens]] | [[Category: Homo sapiens]] | ||
[[Category: Receptor protein-tyrosine kinase]] | [[Category: Receptor protein-tyrosine kinase]] | ||
| - | [[Category: | + | [[Category: Ceska, T.]] |
| - | [[Category: | + | [[Category: Meier, C.]] |
[[Category: Alternative splicing]] | [[Category: Alternative splicing]] | ||
[[Category: Atp-binding]] | [[Category: Atp-binding]] | ||
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[[Category: Tyrosine-protein kinase.]] | [[Category: Tyrosine-protein kinase.]] | ||
| - | ''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on | + | ''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Mon Sep 21 20:14:22 2009'' |
Revision as of 17:14, 21 September 2009
Contents |
Human c-Met Kinase in complex with quinoxaline inhibitor
Template:ABSTRACT PUBMED 19059779
Disease
Known disease associated with this structure: Hepatocellular carcinoma, childhood type OMIM:[164860], Renal cell carcinoma, papillary, familial and sporadic OMIM:[164860], Autism, suseptibility to, 9 OMIM:[164860]
About this Structure
3F66 is a 2 chains structure of sequences from Homo sapiens. Full crystallographic information is available from OCA.
Reference
- Porter J, Lumb S, Lecomte F, Reuberson J, Foley A, Calmiano M, le Riche K, Edwards H, Delgado J, Franklin RJ, Gascon-Simorte JM, Maloney A, Meier C, Batchelor M. Discovery of a novel series of quinoxalines as inhibitors of c-Met kinase. Bioorg Med Chem Lett. 2009 Jan 15;19(2):397-400. Epub 2008 Nov 21. PMID:19059779 doi:10.1016/j.bmcl.2008.11.062
Page seeded by OCA on Mon Sep 21 20:14:22 2009
Categories: Homo sapiens | Receptor protein-tyrosine kinase | Ceska, T. | Meier, C. | Alternative splicing | Atp-binding | C-met | Chromosomal rearrangement | Disease mutation | Glycoprotein | Kinase | Membrane | Nucleotide-binding | Phosphoprotein | Polymorphism | Protein kinase | Proto-oncogene | Quinoxaline | Receptor | Repeat | Signal | Transferase | Transmembrane | Tyrosine-protein kinase.
