1b0q

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(New page: 200px<br /><applet load="1b0q" size="450" color="white" frame="true" align="right" spinBox="true" caption="1b0q" /> '''DITHIOL ALPHA MELANOTROPIN PEPTIDE CYCLIZED ...)
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'''DITHIOL ALPHA MELANOTROPIN PEPTIDE CYCLIZED VIA RHENIUM METAL COORDINATION'''<br />
'''DITHIOL ALPHA MELANOTROPIN PEPTIDE CYCLIZED VIA RHENIUM METAL COORDINATION'''<br />
==Overview==
==Overview==
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alpha-Melanocyte stimulating hormone (alpha-MSH) analogs, cyclized through, site-specific rhenium (Re) and technetium (Tc) metal coordination, were, structurally characterized and analyzed for their abilities to bind, alpha-MSH receptors present on melanoma cells and in tumor-bearing mice., Results from receptor-binding assays conducted with B16 F1 murine melanoma, cells indicated that receptor-binding affinity was reduced to, approximately 1% of its original levels after Re incorporation into the, cyclic Cys4,10, D-Phe7-alpha-MSH4-13 analog. Structural analysis of the, Re-peptide complex showed that the disulfide bond of the original peptide, was replaced by thiolate-metal-thiolate cyclization. A comparison of the, metal-bound and metal-free structures indicated that metal complexation, dramatically altered the structure of the receptor-binding core sequence., Redesign of the metal binding site resulted in a second-generation, Re-peptide complex (ReCCMSH) that displayed a receptor-binding affinity of, 2.9 nM, 25-fold higher than the initial Re-alpha-MSH analog., Characterization of the second-generation Re-peptide complex indicated, that the peptide was still cyclized through Re coordination, but the, structure of the receptor-binding sequence was no longer constrained. The, corresponding 99mTc- and 188ReCCMSH complexes were synthesized and shown, to be stable in phosphate-buffered saline and to challenges from, diethylenetriaminepentaacetic acid (DTPA) and free cysteine. In vivo, the, 99mTcCCMSH complex exhibited significant tumor uptake and retention and, was effective in imaging melanoma in a murine-tumor model system., Cyclization of alpha-MSH analogs via 99mTc and 188Re yields chemically, stable and biologically active molecules with potential melanoma-imaging, and therapeutic properties.
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alpha-Melanocyte stimulating hormone (alpha-MSH) analogs, cyclized through site-specific rhenium (Re) and technetium (Tc) metal coordination, were structurally characterized and analyzed for their abilities to bind alpha-MSH receptors present on melanoma cells and in tumor-bearing mice. Results from receptor-binding assays conducted with B16 F1 murine melanoma cells indicated that receptor-binding affinity was reduced to approximately 1% of its original levels after Re incorporation into the cyclic Cys4,10, D-Phe7-alpha-MSH4-13 analog. Structural analysis of the Re-peptide complex showed that the disulfide bond of the original peptide was replaced by thiolate-metal-thiolate cyclization. A comparison of the metal-bound and metal-free structures indicated that metal complexation dramatically altered the structure of the receptor-binding core sequence. Redesign of the metal binding site resulted in a second-generation Re-peptide complex (ReCCMSH) that displayed a receptor-binding affinity of 2.9 nM, 25-fold higher than the initial Re-alpha-MSH analog. Characterization of the second-generation Re-peptide complex indicated that the peptide was still cyclized through Re coordination, but the structure of the receptor-binding sequence was no longer constrained. The corresponding 99mTc- and 188ReCCMSH complexes were synthesized and shown to be stable in phosphate-buffered saline and to challenges from diethylenetriaminepentaacetic acid (DTPA) and free cysteine. In vivo, the 99mTcCCMSH complex exhibited significant tumor uptake and retention and was effective in imaging melanoma in a murine-tumor model system. Cyclization of alpha-MSH analogs via 99mTc and 188Re yields chemically stable and biologically active molecules with potential melanoma-imaging and therapeutic properties.
==About this Structure==
==About this Structure==
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1B0Q is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/ ] with RE, ACE and NH2 as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1B0Q OCA].
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1B0Q is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/ ] with <scene name='pdbligand=RE:'>RE</scene>, <scene name='pdbligand=ACE:'>ACE</scene> and <scene name='pdbligand=NH2:'>NH2</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1B0Q OCA].
==Reference==
==Reference==
Design and characterization of alpha-melanotropin peptide analogs cyclized through rhenium and technetium metal coordination., Giblin MF, Wang N, Hoffman TJ, Jurisson SS, Quinn TP, Proc Natl Acad Sci U S A. 1998 Oct 27;95(22):12814-8. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=9788997 9788997]
Design and characterization of alpha-melanotropin peptide analogs cyclized through rhenium and technetium metal coordination., Giblin MF, Wang N, Hoffman TJ, Jurisson SS, Quinn TP, Proc Natl Acad Sci U S A. 1998 Oct 27;95(22):12814-8. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=9788997 9788997]
[[Category: Protein complex]]
[[Category: Protein complex]]
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[[Category: Giblin, M.F.]]
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[[Category: Giblin, M F.]]
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[[Category: Hoffman, T.J.]]
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[[Category: Hoffman, T J.]]
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[[Category: Jurisson, S.S.]]
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[[Category: Jurisson, S S.]]
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[[Category: Quinn, T.P.]]
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[[Category: Quinn, T P.]]
[[Category: Wang, N.]]
[[Category: Wang, N.]]
[[Category: ACE]]
[[Category: ACE]]
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[[Category: rhenium technetium]]
[[Category: rhenium technetium]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 11:50:20 2008''

Revision as of 09:50, 21 February 2008


1b0q

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DITHIOL ALPHA MELANOTROPIN PEPTIDE CYCLIZED VIA RHENIUM METAL COORDINATION

Overview

alpha-Melanocyte stimulating hormone (alpha-MSH) analogs, cyclized through site-specific rhenium (Re) and technetium (Tc) metal coordination, were structurally characterized and analyzed for their abilities to bind alpha-MSH receptors present on melanoma cells and in tumor-bearing mice. Results from receptor-binding assays conducted with B16 F1 murine melanoma cells indicated that receptor-binding affinity was reduced to approximately 1% of its original levels after Re incorporation into the cyclic Cys4,10, D-Phe7-alpha-MSH4-13 analog. Structural analysis of the Re-peptide complex showed that the disulfide bond of the original peptide was replaced by thiolate-metal-thiolate cyclization. A comparison of the metal-bound and metal-free structures indicated that metal complexation dramatically altered the structure of the receptor-binding core sequence. Redesign of the metal binding site resulted in a second-generation Re-peptide complex (ReCCMSH) that displayed a receptor-binding affinity of 2.9 nM, 25-fold higher than the initial Re-alpha-MSH analog. Characterization of the second-generation Re-peptide complex indicated that the peptide was still cyclized through Re coordination, but the structure of the receptor-binding sequence was no longer constrained. The corresponding 99mTc- and 188ReCCMSH complexes were synthesized and shown to be stable in phosphate-buffered saline and to challenges from diethylenetriaminepentaacetic acid (DTPA) and free cysteine. In vivo, the 99mTcCCMSH complex exhibited significant tumor uptake and retention and was effective in imaging melanoma in a murine-tumor model system. Cyclization of alpha-MSH analogs via 99mTc and 188Re yields chemically stable and biologically active molecules with potential melanoma-imaging and therapeutic properties.

About this Structure

1B0Q is a Protein complex structure of sequences from [1] with , and as ligands. Full crystallographic information is available from OCA.

Reference

Design and characterization of alpha-melanotropin peptide analogs cyclized through rhenium and technetium metal coordination., Giblin MF, Wang N, Hoffman TJ, Jurisson SS, Quinn TP, Proc Natl Acad Sci U S A. 1998 Oct 27;95(22):12814-8. PMID:9788997

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