1fby

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(New page: 200px<br /> <applet load="1fby" size="450" color="white" frame="true" align="right" spinBox="true" caption="1fby, resolution 2.25&Aring;" /> '''CRYSTAL STRUCTURE O...)
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<applet load="1fby" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="1fby, resolution 2.25&Aring;" />
'''CRYSTAL STRUCTURE OF THE HUMAN RXR ALPHA LIGAND BINDING DOMAIN BOUND TO 9-CIS RETINOIC ACID'''<br />
'''CRYSTAL STRUCTURE OF THE HUMAN RXR ALPHA LIGAND BINDING DOMAIN BOUND TO 9-CIS RETINOIC ACID'''<br />
==Overview==
==Overview==
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The pleiotropic effects of active retinoids are transduced by their, cognate nuclear receptors, retinoid X receptors (RXRs) and retinoic acid, receptors (RARs), which act as transcriptional regulators activated by two, stereoisomers of retinoic acid (RA): 9-cis RA (9-cRA) and all-trans RA, (a-tRA). Among nuclear receptors, RXR occupies a central position and, plays a crucial role in many intracellular signalling pathways as a, ubiquitous heterodimerization partner with numerous other members of this, superfamily. Whereas RARs bind both isomers, RXRs exclusively bind 9-cRA., The crystal structure of the ligand-binding domain (LBD) of human RXRalpha, bound to 9-cRA reveals the molecular basis of this ligand selectivity and, allows a comparison of both apo and holo forms of the same nuclear, receptor. In the crystal, the receptor is monomeric and exhibits a, canonical agonist conformation without direct contacts between the ligand, and the transactivation helix H12. Comparison with the unliganded RXRalpha, LBD structure reveals the molecular mechanisms of ligand-induced, conformational changes and allows us to describe at the atomic level how, these changes generate the proper protein interface involved in nuclear, receptor-coactivator interaction.
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The pleiotropic effects of active retinoids are transduced by their cognate nuclear receptors, retinoid X receptors (RXRs) and retinoic acid receptors (RARs), which act as transcriptional regulators activated by two stereoisomers of retinoic acid (RA): 9-cis RA (9-cRA) and all-trans RA (a-tRA). Among nuclear receptors, RXR occupies a central position and plays a crucial role in many intracellular signalling pathways as a ubiquitous heterodimerization partner with numerous other members of this superfamily. Whereas RARs bind both isomers, RXRs exclusively bind 9-cRA. The crystal structure of the ligand-binding domain (LBD) of human RXRalpha bound to 9-cRA reveals the molecular basis of this ligand selectivity and allows a comparison of both apo and holo forms of the same nuclear receptor. In the crystal, the receptor is monomeric and exhibits a canonical agonist conformation without direct contacts between the ligand and the transactivation helix H12. Comparison with the unliganded RXRalpha LBD structure reveals the molecular mechanisms of ligand-induced conformational changes and allows us to describe at the atomic level how these changes generate the proper protein interface involved in nuclear receptor-coactivator interaction.
==About this Structure==
==About this Structure==
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1FBY is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with REA as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1FBY OCA].
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1FBY is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=REA:'>REA</scene> as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1FBY OCA].
==Reference==
==Reference==
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[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Chambon, P.]]
[[Category: Chambon, P.]]
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[[Category: Egea, P.F.]]
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[[Category: Egea, P F.]]
[[Category: Mitschler, A.]]
[[Category: Mitschler, A.]]
[[Category: Moras, D.]]
[[Category: Moras, D.]]
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[[Category: retinoid receptor]]
[[Category: retinoid receptor]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 16:51:12 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 12:37:11 2008''

Revision as of 10:37, 21 February 2008


1fby, resolution 2.25Å

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CRYSTAL STRUCTURE OF THE HUMAN RXR ALPHA LIGAND BINDING DOMAIN BOUND TO 9-CIS RETINOIC ACID

Overview

The pleiotropic effects of active retinoids are transduced by their cognate nuclear receptors, retinoid X receptors (RXRs) and retinoic acid receptors (RARs), which act as transcriptional regulators activated by two stereoisomers of retinoic acid (RA): 9-cis RA (9-cRA) and all-trans RA (a-tRA). Among nuclear receptors, RXR occupies a central position and plays a crucial role in many intracellular signalling pathways as a ubiquitous heterodimerization partner with numerous other members of this superfamily. Whereas RARs bind both isomers, RXRs exclusively bind 9-cRA. The crystal structure of the ligand-binding domain (LBD) of human RXRalpha bound to 9-cRA reveals the molecular basis of this ligand selectivity and allows a comparison of both apo and holo forms of the same nuclear receptor. In the crystal, the receptor is monomeric and exhibits a canonical agonist conformation without direct contacts between the ligand and the transactivation helix H12. Comparison with the unliganded RXRalpha LBD structure reveals the molecular mechanisms of ligand-induced conformational changes and allows us to describe at the atomic level how these changes generate the proper protein interface involved in nuclear receptor-coactivator interaction.

About this Structure

1FBY is a Single protein structure of sequence from Homo sapiens with as ligand. Full crystallographic information is available from OCA.

Reference

Crystal structure of the human RXRalpha ligand-binding domain bound to its natural ligand: 9-cis retinoic acid., Egea PF, Mitschler A, Rochel N, Ruff M, Chambon P, Moras D, EMBO J. 2000 Jun 1;19(11):2592-601. PMID:10835357

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