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1iml

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(New page: 200px<br /><applet load="1iml" size="450" color="white" frame="true" align="right" spinBox="true" caption="1iml" /> '''CYSTEINE RICH INTESTINAL PROTEIN, NMR, 48 ST...)
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[[Image:1iml.gif|left|200px]]<br /><applet load="1iml" size="350" color="white" frame="true" align="right" spinBox="true"
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'''CYSTEINE RICH INTESTINAL PROTEIN, NMR, 48 STRUCTURES'''<br />
'''CYSTEINE RICH INTESTINAL PROTEIN, NMR, 48 STRUCTURES'''<br />
==Overview==
==Overview==
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LIM domains are Zn-binding arrays found in a number of proteins involved, in the control of cell differentiation, including several developmentally, regulated transcription factors and a human proto-oncogene product. The, rat cysteine-rich intestinal protein, CRIP, is a 76-residue polypeptide, which contains a LIM motif. The solution structure of CRIP has been, determined by homonuclear and 1H-15N heteronuclear correlated nuclear, magnetic resonance spectroscopy. Structures with individual distance, violations of &lt; or = 0.03 angstrom and penalties (squared sum of distance, violations) of &lt; or = 0.06 angstrom2 were generated with a total of 500, nuclear Overhauser effect (NOE)-derived distance restraints (averaging, 15.6 restraints per refined residue). Superposition of backbone heavy, atoms of ordered residues relative to mean atom positions is achieved with, pairwise rms deviations of 0.54(+/-0.14) angstrom. As observed previously, for a peptide with the sequence of the C-terminal LIM domain from the, avian cysteine-rich protein, CRP (cCRP-LIM2), CRIP binds two equivalents, of zinc, forming N-terminal CCHC (Cys3, Cys6, His24, Cys27) and C-terminal, CCCC (Cys30, Cys33, Cys51, Cys55) modules. The CCHC and CCCC modules in, CRIP contain two orthogonally-arrayed antiparallel beta-sheets. The, C-terminal end of the CCHC module contains a tight turn and the C terminus, of the CCCC module forms an alpha-helix. The modules pack via hydrophobic, interactions, forming a compact structure that is similar to that observed, for cCRP-LIM2. The most significant differences between the structures, occur at the CCHC module-CCCC module interface, which results in a, difference in the relative orientations of the modules, and at the C, terminus where the alpha-helix appears to be packed more tightly against, the preceding antiparallel beta-sheet. The greater abundance of NOE, information obtained for CRIP relative to cCRP-LIM2, combined with the, analysis of J-coupling and proton chemical shift data, have allowed a more, detailed evaluation of the molecular level interactions that stabilize the, fold of the LIM motif.
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LIM domains are Zn-binding arrays found in a number of proteins involved in the control of cell differentiation, including several developmentally regulated transcription factors and a human proto-oncogene product. The rat cysteine-rich intestinal protein, CRIP, is a 76-residue polypeptide which contains a LIM motif. The solution structure of CRIP has been determined by homonuclear and 1H-15N heteronuclear correlated nuclear magnetic resonance spectroscopy. Structures with individual distance violations of &lt; or = 0.03 angstrom and penalties (squared sum of distance violations) of &lt; or = 0.06 angstrom2 were generated with a total of 500 nuclear Overhauser effect (NOE)-derived distance restraints (averaging 15.6 restraints per refined residue). Superposition of backbone heavy atoms of ordered residues relative to mean atom positions is achieved with pairwise rms deviations of 0.54(+/-0.14) angstrom. As observed previously for a peptide with the sequence of the C-terminal LIM domain from the avian cysteine-rich protein, CRP (cCRP-LIM2), CRIP binds two equivalents of zinc, forming N-terminal CCHC (Cys3, Cys6, His24, Cys27) and C-terminal CCCC (Cys30, Cys33, Cys51, Cys55) modules. The CCHC and CCCC modules in CRIP contain two orthogonally-arrayed antiparallel beta-sheets. The C-terminal end of the CCHC module contains a tight turn and the C terminus of the CCCC module forms an alpha-helix. The modules pack via hydrophobic interactions, forming a compact structure that is similar to that observed for cCRP-LIM2. The most significant differences between the structures occur at the CCHC module-CCCC module interface, which results in a difference in the relative orientations of the modules, and at the C terminus where the alpha-helix appears to be packed more tightly against the preceding antiparallel beta-sheet. The greater abundance of NOE information obtained for CRIP relative to cCRP-LIM2, combined with the analysis of J-coupling and proton chemical shift data, have allowed a more detailed evaluation of the molecular level interactions that stabilize the fold of the LIM motif.
==About this Structure==
==About this Structure==
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1IML is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Rattus_rattus Rattus rattus] with ZN as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1IML OCA].
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1IML is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Rattus_rattus Rattus rattus] with <scene name='pdbligand=ZN:'>ZN</scene> as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1IML OCA].
==Reference==
==Reference==
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[[Category: Rattus rattus]]
[[Category: Rattus rattus]]
[[Category: Single protein]]
[[Category: Single protein]]
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[[Category: Beckerle, M.C.]]
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[[Category: Beckerle, M C.]]
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[[Category: Kosa, J.L.]]
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[[Category: Kosa, J L.]]
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[[Category: Louis, H.A.]]
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[[Category: Louis, H A.]]
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[[Category: Perez-Alvarado, G.C.]]
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[[Category: Perez-Alvarado, G C.]]
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[[Category: Summers, M.F.]]
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[[Category: Summers, M F.]]
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[[Category: Winge, D.R.]]
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[[Category: Winge, D R.]]
[[Category: ZN]]
[[Category: ZN]]
[[Category: lim domain protein]]
[[Category: lim domain protein]]
[[Category: metal-binding protein]]
[[Category: metal-binding protein]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Tue Nov 20 17:30:21 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 13:13:22 2008''

Revision as of 11:13, 21 February 2008


1iml

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CYSTEINE RICH INTESTINAL PROTEIN, NMR, 48 STRUCTURES

Overview

LIM domains are Zn-binding arrays found in a number of proteins involved in the control of cell differentiation, including several developmentally regulated transcription factors and a human proto-oncogene product. The rat cysteine-rich intestinal protein, CRIP, is a 76-residue polypeptide which contains a LIM motif. The solution structure of CRIP has been determined by homonuclear and 1H-15N heteronuclear correlated nuclear magnetic resonance spectroscopy. Structures with individual distance violations of < or = 0.03 angstrom and penalties (squared sum of distance violations) of < or = 0.06 angstrom2 were generated with a total of 500 nuclear Overhauser effect (NOE)-derived distance restraints (averaging 15.6 restraints per refined residue). Superposition of backbone heavy atoms of ordered residues relative to mean atom positions is achieved with pairwise rms deviations of 0.54(+/-0.14) angstrom. As observed previously for a peptide with the sequence of the C-terminal LIM domain from the avian cysteine-rich protein, CRP (cCRP-LIM2), CRIP binds two equivalents of zinc, forming N-terminal CCHC (Cys3, Cys6, His24, Cys27) and C-terminal CCCC (Cys30, Cys33, Cys51, Cys55) modules. The CCHC and CCCC modules in CRIP contain two orthogonally-arrayed antiparallel beta-sheets. The C-terminal end of the CCHC module contains a tight turn and the C terminus of the CCCC module forms an alpha-helix. The modules pack via hydrophobic interactions, forming a compact structure that is similar to that observed for cCRP-LIM2. The most significant differences between the structures occur at the CCHC module-CCCC module interface, which results in a difference in the relative orientations of the modules, and at the C terminus where the alpha-helix appears to be packed more tightly against the preceding antiparallel beta-sheet. The greater abundance of NOE information obtained for CRIP relative to cCRP-LIM2, combined with the analysis of J-coupling and proton chemical shift data, have allowed a more detailed evaluation of the molecular level interactions that stabilize the fold of the LIM motif.

About this Structure

1IML is a Single protein structure of sequence from Rattus rattus with as ligand. Full crystallographic information is available from OCA.

Reference

Structure of the cysteine-rich intestinal protein, CRIP., Perez-Alvarado GC, Kosa JL, Louis HA, Beckerle MC, Winge DR, Summers MF, J Mol Biol. 1996 Mar 22;257(1):153-74. PMID:8632452

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