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1k1f

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(New page: 200px<br /> <applet load="1k1f" size="450" color="white" frame="true" align="right" spinBox="true" caption="1k1f, resolution 2.20&Aring;" /> '''Structure of the Bc...)
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[[Image:1k1f.gif|left|200px]]<br />
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[[Image:1k1f.gif|left|200px]]<br /><applet load="1k1f" size="350" color="white" frame="true" align="right" spinBox="true"
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<applet load="1k1f" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="1k1f, resolution 2.20&Aring;" />
'''Structure of the Bcr-Abl Oncoprotein Oligomerization domain'''<br />
'''Structure of the Bcr-Abl Oncoprotein Oligomerization domain'''<br />
==Overview==
==Overview==
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The Bcr-Abl oncoprotein is responsible for a wide range of human, leukemias, including most cases of Philadelphia chromosome-positive, chronic myelogenous leukemia. Oligomerization of Bcr-Abl is essential for, oncogenicity. We determined the crystal structure of the N-terminal, oligomerization domain of Bcr-Abl (residues 1-72 or Bcr1-72) and found a, novel mode of oligomer formation. Two N-shaped monomers dimerize by, swapping N-terminal helices and by forming an antiparallel coiled coil, between C-terminal helices. Two dimers then stack onto each other to form, a tetramer. The Bcr1-72 structure provides a basis for the design of, inhibitors of Bcr-Abl transforming activity by disrupting Bcr-Abl, oligomerization.
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The Bcr-Abl oncoprotein is responsible for a wide range of human leukemias, including most cases of Philadelphia chromosome-positive chronic myelogenous leukemia. Oligomerization of Bcr-Abl is essential for oncogenicity. We determined the crystal structure of the N-terminal oligomerization domain of Bcr-Abl (residues 1-72 or Bcr1-72) and found a novel mode of oligomer formation. Two N-shaped monomers dimerize by swapping N-terminal helices and by forming an antiparallel coiled coil between C-terminal helices. Two dimers then stack onto each other to form a tetramer. The Bcr1-72 structure provides a basis for the design of inhibitors of Bcr-Abl transforming activity by disrupting Bcr-Abl oligomerization.
==Disease==
==Disease==
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==About this Structure==
==About this Structure==
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1K1F is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1K1F OCA].
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1K1F is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1K1F OCA].
==Reference==
==Reference==
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[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Ghaffari, S.]]
[[Category: Ghaffari, S.]]
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[[Category: Kim, P.S.]]
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[[Category: Kim, P S.]]
[[Category: Lodish, H.]]
[[Category: Lodish, H.]]
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[[Category: Malashkevich, V.N.]]
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[[Category: Malashkevich, V N.]]
[[Category: Zhao, X.]]
[[Category: Zhao, X.]]
[[Category: bcr-abl kinase]]
[[Category: bcr-abl kinase]]
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[[Category: oligomerization]]
[[Category: oligomerization]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 17:46:31 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 13:29:13 2008''

Revision as of 11:29, 21 February 2008


1k1f, resolution 2.20Å

Drag the structure with the mouse to rotate

Structure of the Bcr-Abl Oncoprotein Oligomerization domain

Contents

Overview

The Bcr-Abl oncoprotein is responsible for a wide range of human leukemias, including most cases of Philadelphia chromosome-positive chronic myelogenous leukemia. Oligomerization of Bcr-Abl is essential for oncogenicity. We determined the crystal structure of the N-terminal oligomerization domain of Bcr-Abl (residues 1-72 or Bcr1-72) and found a novel mode of oligomer formation. Two N-shaped monomers dimerize by swapping N-terminal helices and by forming an antiparallel coiled coil between C-terminal helices. Two dimers then stack onto each other to form a tetramer. The Bcr1-72 structure provides a basis for the design of inhibitors of Bcr-Abl transforming activity by disrupting Bcr-Abl oligomerization.

Disease

Known diseases associated with this structure: Leukemia, acute lymphocytic OMIM:[151410], Leukemia, chronic myeloid OMIM:[151410]

About this Structure

1K1F is a Single protein structure of sequence from Homo sapiens. Full crystallographic information is available from OCA.

Reference

Structure of the Bcr-Abl oncoprotein oligomerization domain., Zhao X, Ghaffari S, Lodish H, Malashkevich VN, Kim PS, Nat Struct Biol. 2002 Feb;9(2):117-20. PMID:11780146

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