1mh0

From Proteopedia

(Difference between revisions)
Jump to: navigation, search
(New page: 200px<br /> <applet load="1mh0" size="450" color="white" frame="true" align="right" spinBox="true" caption="1mh0, resolution 2.80&Aring;" /> '''Crystal structure o...)
Line 1: Line 1:
-
[[Image:1mh0.gif|left|200px]]<br />
+
[[Image:1mh0.gif|left|200px]]<br /><applet load="1mh0" size="350" color="white" frame="true" align="right" spinBox="true"
-
<applet load="1mh0" size="450" color="white" frame="true" align="right" spinBox="true"
+
caption="1mh0, resolution 2.80&Aring;" />
caption="1mh0, resolution 2.80&Aring;" />
'''Crystal structure of the anticoagulant slow form of thrombin'''<br />
'''Crystal structure of the anticoagulant slow form of thrombin'''<br />
==Overview==
==Overview==
-
Using the thrombin mutant R77aA devoid of the site of autoproteolytic, degradation at exosite I, we have solved for the first time the structure, of thrombin free of any inhibitors and effector molecules and stabilized, in the Na(+)-free slow form. The slow form shows subtle differences, compared with the currently available structures of the Na(+)-bound fast, form that carry inhibitors at the active site or exosite I. The most, notable differences are the displacement of Asp-189 in the S1 specificity, pocket, a downward shift of the 190-193 strand, a rearrangement of the, side chain of Glu-192, and a significant shift in the position of the, catalytic Ser-195 that is no longer within H-bonding distance from His-57., The structure of the slow form explains the reduced specificity toward, synthetic and natural substrates and suggests a molecular basis for its, anticoagulant properties.
+
Using the thrombin mutant R77aA devoid of the site of autoproteolytic degradation at exosite I, we have solved for the first time the structure of thrombin free of any inhibitors and effector molecules and stabilized in the Na(+)-free slow form. The slow form shows subtle differences compared with the currently available structures of the Na(+)-bound fast form that carry inhibitors at the active site or exosite I. The most notable differences are the displacement of Asp-189 in the S1 specificity pocket, a downward shift of the 190-193 strand, a rearrangement of the side chain of Glu-192, and a significant shift in the position of the catalytic Ser-195 that is no longer within H-bonding distance from His-57. The structure of the slow form explains the reduced specificity toward synthetic and natural substrates and suggests a molecular basis for its anticoagulant properties.
==Disease==
==Disease==
Line 11: Line 10:
==About this Structure==
==About this Structure==
-
1MH0 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with NAG as [http://en.wikipedia.org/wiki/ligand ligand]. Active as [http://en.wikipedia.org/wiki/Thrombin Thrombin], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.4.21.5 3.4.21.5] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1MH0 OCA].
+
1MH0 is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=NAG:'>NAG</scene> as [http://en.wikipedia.org/wiki/ligand ligand]. Active as [http://en.wikipedia.org/wiki/Thrombin Thrombin], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=3.4.21.5 3.4.21.5] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1MH0 OCA].
==Reference==
==Reference==
Line 18: Line 17:
[[Category: Single protein]]
[[Category: Single protein]]
[[Category: Thrombin]]
[[Category: Thrombin]]
-
[[Category: Cera, E.Di.]]
+
[[Category: Cera, E Di.]]
-
[[Category: Pineda, A.O.]]
+
[[Category: Pineda, A O.]]
[[Category: Savvides, S.]]
[[Category: Savvides, S.]]
[[Category: Waksman, G.]]
[[Category: Waksman, G.]]
Line 28: Line 27:
[[Category: thrombin]]
[[Category: thrombin]]
-
''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 18:11:06 2007''
+
''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 13:55:05 2008''

Revision as of 11:55, 21 February 2008


1mh0, resolution 2.80Å

Drag the structure with the mouse to rotate

Crystal structure of the anticoagulant slow form of thrombin

Contents

Overview

Using the thrombin mutant R77aA devoid of the site of autoproteolytic degradation at exosite I, we have solved for the first time the structure of thrombin free of any inhibitors and effector molecules and stabilized in the Na(+)-free slow form. The slow form shows subtle differences compared with the currently available structures of the Na(+)-bound fast form that carry inhibitors at the active site or exosite I. The most notable differences are the displacement of Asp-189 in the S1 specificity pocket, a downward shift of the 190-193 strand, a rearrangement of the side chain of Glu-192, and a significant shift in the position of the catalytic Ser-195 that is no longer within H-bonding distance from His-57. The structure of the slow form explains the reduced specificity toward synthetic and natural substrates and suggests a molecular basis for its anticoagulant properties.

Disease

Known diseases associated with this structure: Dysprothrombinemia OMIM:[176930], Hyperprothrombinemia OMIM:[176930], Hypoprothrombinemia OMIM:[176930]

About this Structure

1MH0 is a Single protein structure of sequence from Homo sapiens with as ligand. Active as Thrombin, with EC number 3.4.21.5 Full crystallographic information is available from OCA.

Reference

Crystal structure of the anticoagulant slow form of thrombin., Pineda AO, Savvides SN, Waksman G, Di Cera E, J Biol Chem. 2002 Oct 25;277(43):40177-80. Epub 2002 Aug 29. PMID:12205081

Page seeded by OCA on Thu Feb 21 13:55:05 2008

Proteopedia Page Contributors and Editors (what is this?)

OCA

Personal tools