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1qz7

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(New page: 200px<br /> <applet load="1qz7" size="450" color="white" frame="true" align="right" spinBox="true" caption="1qz7, resolution 2.20&Aring;" /> '''Beta-catenin bindin...)
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[[Image:1qz7.gif|left|200px]]<br /><applet load="1qz7" size="350" color="white" frame="true" align="right" spinBox="true"
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<applet load="1qz7" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="1qz7, resolution 2.20&Aring;" />
'''Beta-catenin binding domain of Axin in complex with beta-catenin'''<br />
'''Beta-catenin binding domain of Axin in complex with beta-catenin'''<br />
==Overview==
==Overview==
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The "beta-catenin destruction complex" is central to canonical, Wnt/beta-catenin signaling. The scaffolding protein Axin and the tumor, suppressor adenomatous polyposis coli protein (APC) are critical, components of this complex, required for rapid beta-catenin turnover. We, determined the crystal structure of a complex between beta-catenin and the, beta-catenin-binding domain of Axin (Axin-CBD). The Axin-CBD forms a helix, that occupies the groove formed by the third and fourth armadillo repeats, of beta-catenin and thus precludes the simultaneous binding of other, beta-catenin partners in this region. Our biochemical studies demonstrate, that, when phosphorylated, the 20-amino acid repeat region of APC competes, with Axin for binding to beta-catenin. We propose that a key function of, APC in the beta-catenin destruction complex is to remove phosphorylated, beta-catenin product from the active site.
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The "beta-catenin destruction complex" is central to canonical Wnt/beta-catenin signaling. The scaffolding protein Axin and the tumor suppressor adenomatous polyposis coli protein (APC) are critical components of this complex, required for rapid beta-catenin turnover. We determined the crystal structure of a complex between beta-catenin and the beta-catenin-binding domain of Axin (Axin-CBD). The Axin-CBD forms a helix that occupies the groove formed by the third and fourth armadillo repeats of beta-catenin and thus precludes the simultaneous binding of other beta-catenin partners in this region. Our biochemical studies demonstrate that, when phosphorylated, the 20-amino acid repeat region of APC competes with Axin for binding to beta-catenin. We propose that a key function of APC in the beta-catenin destruction complex is to remove phosphorylated beta-catenin product from the active site.
==Disease==
==Disease==
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==About this Structure==
==About this Structure==
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1QZ7 is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] and [http://en.wikipedia.org/wiki/Xenopus_laevis Xenopus laevis]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1QZ7 OCA].
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1QZ7 is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] and [http://en.wikipedia.org/wiki/Xenopus_laevis Xenopus laevis]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1QZ7 OCA].
==Reference==
==Reference==
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[[Category: Protein complex]]
[[Category: Protein complex]]
[[Category: Xenopus laevis]]
[[Category: Xenopus laevis]]
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[[Category: Clements, W.K.]]
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[[Category: Clements, W K.]]
[[Category: Kimelman, D.]]
[[Category: Kimelman, D.]]
[[Category: Xing, Y.]]
[[Category: Xing, Y.]]
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[[Category: protein-protein complex]]
[[Category: protein-protein complex]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 18:57:46 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 14:45:19 2008''

Revision as of 12:45, 21 February 2008


1qz7, resolution 2.20Å

Drag the structure with the mouse to rotate

Beta-catenin binding domain of Axin in complex with beta-catenin

Contents

Overview

The "beta-catenin destruction complex" is central to canonical Wnt/beta-catenin signaling. The scaffolding protein Axin and the tumor suppressor adenomatous polyposis coli protein (APC) are critical components of this complex, required for rapid beta-catenin turnover. We determined the crystal structure of a complex between beta-catenin and the beta-catenin-binding domain of Axin (Axin-CBD). The Axin-CBD forms a helix that occupies the groove formed by the third and fourth armadillo repeats of beta-catenin and thus precludes the simultaneous binding of other beta-catenin partners in this region. Our biochemical studies demonstrate that, when phosphorylated, the 20-amino acid repeat region of APC competes with Axin for binding to beta-catenin. We propose that a key function of APC in the beta-catenin destruction complex is to remove phosphorylated beta-catenin product from the active site.

Disease

Known diseases associated with this structure: Colorectal cancer OMIM:[116806], Hepatoblastoma OMIM:[116806], Hepatocellular carcinoma OMIM:[116806], Ovarian carcinoma, endometrioid type OMIM:[116806], Pilomatricoma OMIM:[116806]

About this Structure

1QZ7 is a Protein complex structure of sequences from Homo sapiens and Xenopus laevis. Full crystallographic information is available from OCA.

Reference

Crystal structure of a beta-catenin/axin complex suggests a mechanism for the beta-catenin destruction complex., Xing Y, Clements WK, Kimelman D, Xu W, Genes Dev. 2003 Nov 15;17(22):2753-64. Epub 2003 Nov 4. PMID:14600025

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