1r6f

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(New page: 200px<br /><applet load="1r6f" size="450" color="white" frame="true" align="right" spinBox="true" caption="1r6f, resolution 2.17&Aring;" /> '''The structure of Yer...)
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[[Image:1r6f.jpg|left|200px]]<br /><applet load="1r6f" size="350" color="white" frame="true" align="right" spinBox="true"
caption="1r6f, resolution 2.17&Aring;" />
caption="1r6f, resolution 2.17&Aring;" />
'''The structure of Yersinia pestis V-antigen, an essential virulence factor and mediator of immunity against plague'''<br />
'''The structure of Yersinia pestis V-antigen, an essential virulence factor and mediator of immunity against plague'''<br />
==Overview==
==Overview==
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The LcrV protein (V-antigen) is a multifunctional virulence factor in, Yersinia pestis, the causative agent of plague. LcrV regulates the, translocation of cytotoxic effector proteins from the bacterium into the, cytosol of mammalian cells via a type III secretion system, possesses, antihost activities of its own, and is also an active and passive mediator, of resistance to disease. Although a crystal structure of this protein has, been actively sought for better understanding of its role in pathogenesis, the wild-type LcrV was found to be recalcitrant to crystallization. We, employed a surface entropy reduction mutagenesis strategy to obtain, crystals of LcrV that diffract to 2.2 A and determined its structure. The, refined model reveals a dumbbell-like molecule with a novel fold that, includes an unexpected coiled-coil motif, and provides a detailed, three-dimensional roadmap for exploring structure-function relationships, in this essential virulence determinant.
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The LcrV protein (V-antigen) is a multifunctional virulence factor in Yersinia pestis, the causative agent of plague. LcrV regulates the translocation of cytotoxic effector proteins from the bacterium into the cytosol of mammalian cells via a type III secretion system, possesses antihost activities of its own, and is also an active and passive mediator of resistance to disease. Although a crystal structure of this protein has been actively sought for better understanding of its role in pathogenesis, the wild-type LcrV was found to be recalcitrant to crystallization. We employed a surface entropy reduction mutagenesis strategy to obtain crystals of LcrV that diffract to 2.2 A and determined its structure. The refined model reveals a dumbbell-like molecule with a novel fold that includes an unexpected coiled-coil motif, and provides a detailed three-dimensional roadmap for exploring structure-function relationships in this essential virulence determinant.
==About this Structure==
==About this Structure==
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1R6F is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Yersinia_pestis Yersinia pestis]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1R6F OCA].
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1R6F is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Yersinia_pestis Yersinia pestis]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1R6F OCA].
==Reference==
==Reference==
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[[Category: Yersinia pestis]]
[[Category: Yersinia pestis]]
[[Category: Derewenda, U.]]
[[Category: Derewenda, U.]]
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[[Category: Derewenda, Z.S.]]
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[[Category: Derewenda, Z S.]]
[[Category: Devedjiev, Y.]]
[[Category: Devedjiev, Y.]]
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[[Category: Evdokimov, A.G.]]
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[[Category: Evdokimov, A G.]]
[[Category: Mateja, A.]]
[[Category: Mateja, A.]]
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[[Category: Routzahn, K.M.]]
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[[Category: Routzahn, K M.]]
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[[Category: Waugh, D.S.]]
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[[Category: Waugh, D S.]]
[[Category: coiled-coil]]
[[Category: coiled-coil]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Wed Nov 21 01:18:27 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 14:47:35 2008''

Revision as of 12:47, 21 February 2008


1r6f, resolution 2.17Å

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The structure of Yersinia pestis V-antigen, an essential virulence factor and mediator of immunity against plague

Overview

The LcrV protein (V-antigen) is a multifunctional virulence factor in Yersinia pestis, the causative agent of plague. LcrV regulates the translocation of cytotoxic effector proteins from the bacterium into the cytosol of mammalian cells via a type III secretion system, possesses antihost activities of its own, and is also an active and passive mediator of resistance to disease. Although a crystal structure of this protein has been actively sought for better understanding of its role in pathogenesis, the wild-type LcrV was found to be recalcitrant to crystallization. We employed a surface entropy reduction mutagenesis strategy to obtain crystals of LcrV that diffract to 2.2 A and determined its structure. The refined model reveals a dumbbell-like molecule with a novel fold that includes an unexpected coiled-coil motif, and provides a detailed three-dimensional roadmap for exploring structure-function relationships in this essential virulence determinant.

About this Structure

1R6F is a Single protein structure of sequence from Yersinia pestis. Full crystallographic information is available from OCA.

Reference

The structure of Yersinia pestis V-antigen, an essential virulence factor and mediator of immunity against plague., Derewenda U, Mateja A, Devedjiev Y, Routzahn KM, Evdokimov AG, Derewenda ZS, Waugh DS, Structure. 2004 Feb;12(2):301-6. PMID:14962390

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