1uxy

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(New page: 200px<br /><applet load="1uxy" size="450" color="white" frame="true" align="right" spinBox="true" caption="1uxy, resolution 1.80&Aring;" /> '''MURB MUTANT WITH SER...)
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[[Image:1uxy.jpg|left|200px]]<br /><applet load="1uxy" size="450" color="white" frame="true" align="right" spinBox="true"
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[[Image:1uxy.jpg|left|200px]]<br /><applet load="1uxy" size="350" color="white" frame="true" align="right" spinBox="true"
caption="1uxy, resolution 1.80&Aring;" />
caption="1uxy, resolution 1.80&Aring;" />
'''MURB MUTANT WITH SER 229 REPLACED BY ALA, COMPLEX WITH ENOLPYRUVYL-UDP-N-ACETYLGLUCOSAMINE'''<br />
'''MURB MUTANT WITH SER 229 REPLACED BY ALA, COMPLEX WITH ENOLPYRUVYL-UDP-N-ACETYLGLUCOSAMINE'''<br />
==Overview==
==Overview==
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MurB catalyzes the second committed step in the synthesis of, peptidoglycan, a key component of the bacterial cell wall. The crystal, structures of both a S229A mutant and wild-type MurB in the presence of, the substrate enolpyruvyl-UDP-N-acetylglucosamine were solved and refined, at 1.8 A resolution. The single point mutation of residue 229 from serine, to alanine eliminated a hydroxyl group which has previously been proposed, to play a critical role as a proton donor during the second half-reaction, of MurB, namely, reoxidation of FADH2 and reduction of the enolpyruvyl, substrate. The mutation also resulted in the loss of the water, molecule-hydrogen bonded to the serine hydroxyl in the wild-type structure, changing the hydrogen-bonding network with in the active site. Comparison, of the wild-type and S229A mutant structures confirms that the dramatic, kinetic defect of an approximately 10(7)-fold decrease observed for the, Ser 229 Ala mutant in the second half-reaction [Benson, T.E., Walsh, C.T., &amp; Massey, V. (1997) Biochemistry 36, 796-805] is a direct result of the, loss of the serine hydroxyl moiety rather than other nonspecific, active-site changes or general structural defects.
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MurB catalyzes the second committed step in the synthesis of peptidoglycan, a key component of the bacterial cell wall. The crystal structures of both a S229A mutant and wild-type MurB in the presence of the substrate enolpyruvyl-UDP-N-acetylglucosamine were solved and refined at 1.8 A resolution. The single point mutation of residue 229 from serine to alanine eliminated a hydroxyl group which has previously been proposed to play a critical role as a proton donor during the second half-reaction of MurB, namely, reoxidation of FADH2 and reduction of the enolpyruvyl substrate. The mutation also resulted in the loss of the water molecule-hydrogen bonded to the serine hydroxyl in the wild-type structure changing the hydrogen-bonding network with in the active site. Comparison of the wild-type and S229A mutant structures confirms that the dramatic kinetic defect of an approximately 10(7)-fold decrease observed for the Ser 229 Ala mutant in the second half-reaction [Benson, T.E., Walsh, C.T., &amp; Massey, V. (1997) Biochemistry 36, 796-805] is a direct result of the loss of the serine hydroxyl moiety rather than other nonspecific active-site changes or general structural defects.
==About this Structure==
==About this Structure==
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1UXY is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Escherichia_coli Escherichia coli] with FAD and EPU as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/UDP-N-acetylmuramate_dehydrogenase UDP-N-acetylmuramate dehydrogenase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=1.1.1.158 1.1.1.158] Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1UXY OCA].
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1UXY is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Escherichia_coli Escherichia coli] with <scene name='pdbligand=FAD:'>FAD</scene> and <scene name='pdbligand=EPU:'>EPU</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Active as [http://en.wikipedia.org/wiki/UDP-N-acetylmuramate_dehydrogenase UDP-N-acetylmuramate dehydrogenase], with EC number [http://www.brenda-enzymes.info/php/result_flat.php4?ecno=1.1.1.158 1.1.1.158] Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1UXY OCA].
==Reference==
==Reference==
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[[Category: Single protein]]
[[Category: Single protein]]
[[Category: UDP-N-acetylmuramate dehydrogenase]]
[[Category: UDP-N-acetylmuramate dehydrogenase]]
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[[Category: Benson, T.E.]]
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[[Category: Benson, T E.]]
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[[Category: Hogle, J.M.]]
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[[Category: Hogle, J M.]]
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[[Category: Walsh, C.T.]]
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[[Category: Walsh, C T.]]
[[Category: EPU]]
[[Category: EPU]]
[[Category: FAD]]
[[Category: FAD]]
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[[Category: peptidoglycan synthesis]]
[[Category: peptidoglycan synthesis]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Wed Nov 21 04:21:25 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 15:29:32 2008''

Revision as of 13:29, 21 February 2008


1uxy, resolution 1.80Å

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MURB MUTANT WITH SER 229 REPLACED BY ALA, COMPLEX WITH ENOLPYRUVYL-UDP-N-ACETYLGLUCOSAMINE

Overview

MurB catalyzes the second committed step in the synthesis of peptidoglycan, a key component of the bacterial cell wall. The crystal structures of both a S229A mutant and wild-type MurB in the presence of the substrate enolpyruvyl-UDP-N-acetylglucosamine were solved and refined at 1.8 A resolution. The single point mutation of residue 229 from serine to alanine eliminated a hydroxyl group which has previously been proposed to play a critical role as a proton donor during the second half-reaction of MurB, namely, reoxidation of FADH2 and reduction of the enolpyruvyl substrate. The mutation also resulted in the loss of the water molecule-hydrogen bonded to the serine hydroxyl in the wild-type structure changing the hydrogen-bonding network with in the active site. Comparison of the wild-type and S229A mutant structures confirms that the dramatic kinetic defect of an approximately 10(7)-fold decrease observed for the Ser 229 Ala mutant in the second half-reaction [Benson, T.E., Walsh, C.T., & Massey, V. (1997) Biochemistry 36, 796-805] is a direct result of the loss of the serine hydroxyl moiety rather than other nonspecific active-site changes or general structural defects.

About this Structure

1UXY is a Single protein structure of sequence from Escherichia coli with and as ligands. Active as UDP-N-acetylmuramate dehydrogenase, with EC number 1.1.1.158 Full crystallographic information is available from OCA.

Reference

X-ray crystal structures of the S229A mutant and wild-type MurB in the presence of the substrate enolpyruvyl-UDP-N-acetylglucosamine at 1.8-A resolution., Benson TE, Walsh CT, Hogle JM, Biochemistry. 1997 Jan 28;36(4):806-11. PMID:9020778

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