1wms

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'''High resolution crystal structure of human Rab9 GTPase: a novel antiviral drug target'''<br />
'''High resolution crystal structure of human Rab9 GTPase: a novel antiviral drug target'''<br />
==Overview==
==Overview==
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Rab GTPases and their effectors facilitate vesicular transport by, tethering donor vesicles to their respective target membranes. Rab9, mediates late endosome to trans-Golgi transport and has recently been, found to be a key cellular component for human immunodeficiency virus-1, Ebola, Marburg, and measles virus replication, suggesting that it may be a, novel target in the development of broad spectrum antiviral drugs. As part, of our structure-based drug design program, we have determined the crystal, structure of a C-terminally truncated human Rab9 (residues 1-177) to, 1.25-A resolution. The overall structure shows a characteristic nucleotide, binding fold consisting of a six-stranded beta-sheet surrounded by five, alpha-helices with a tightly bound GDP molecule in the active site., Structure-based sequence alignment of Rab9 with other Rab proteins reveals, that its active site consists of residues highly conserved in the Rab, GTPase family, implying a common catalytic mechanism. However, Rab9, contains seven regions that are significantly different in conformation, from other Rab proteins. Some of those regions coincide with putative, effector-binding sites and switch I and switch II regions identified by, structure/sequence alignments. The Rab9 structure at near atomic, resolution provides an excellent model for structure-based antiviral drug, design.
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Rab GTPases and their effectors facilitate vesicular transport by tethering donor vesicles to their respective target membranes. Rab9 mediates late endosome to trans-Golgi transport and has recently been found to be a key cellular component for human immunodeficiency virus-1, Ebola, Marburg, and measles virus replication, suggesting that it may be a novel target in the development of broad spectrum antiviral drugs. As part of our structure-based drug design program, we have determined the crystal structure of a C-terminally truncated human Rab9 (residues 1-177) to 1.25-A resolution. The overall structure shows a characteristic nucleotide binding fold consisting of a six-stranded beta-sheet surrounded by five alpha-helices with a tightly bound GDP molecule in the active site. Structure-based sequence alignment of Rab9 with other Rab proteins reveals that its active site consists of residues highly conserved in the Rab GTPase family, implying a common catalytic mechanism. However, Rab9 contains seven regions that are significantly different in conformation from other Rab proteins. Some of those regions coincide with putative effector-binding sites and switch I and switch II regions identified by structure/sequence alignments. The Rab9 structure at near atomic resolution provides an excellent model for structure-based antiviral drug design.
==About this Structure==
==About this Structure==
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1WMS is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with GDP as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=1WMS OCA].
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1WMS is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=GDP:'>GDP</scene> as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=1WMS OCA].
==Reference==
==Reference==
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[[Category: Chen, L.]]
[[Category: Chen, L.]]
[[Category: DiGiammarino, E.]]
[[Category: DiGiammarino, E.]]
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[[Category: Hodge, T.W.]]
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[[Category: Hodge, T W.]]
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[[Category: Meehan, E.J.]]
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[[Category: Meehan, E J.]]
[[Category: Toh, D.]]
[[Category: Toh, D.]]
[[Category: Wang, Y.]]
[[Category: Wang, Y.]]
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[[Category: Zhou, X.E.]]
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[[Category: Zhou, X E.]]
[[Category: GDP]]
[[Category: GDP]]
[[Category: gtpase]]
[[Category: gtpase]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 15:46:02 2008''

Revision as of 13:46, 21 February 2008


1wms, resolution 1.25Å

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High resolution crystal structure of human Rab9 GTPase: a novel antiviral drug target

Overview

Rab GTPases and their effectors facilitate vesicular transport by tethering donor vesicles to their respective target membranes. Rab9 mediates late endosome to trans-Golgi transport and has recently been found to be a key cellular component for human immunodeficiency virus-1, Ebola, Marburg, and measles virus replication, suggesting that it may be a novel target in the development of broad spectrum antiviral drugs. As part of our structure-based drug design program, we have determined the crystal structure of a C-terminally truncated human Rab9 (residues 1-177) to 1.25-A resolution. The overall structure shows a characteristic nucleotide binding fold consisting of a six-stranded beta-sheet surrounded by five alpha-helices with a tightly bound GDP molecule in the active site. Structure-based sequence alignment of Rab9 with other Rab proteins reveals that its active site consists of residues highly conserved in the Rab GTPase family, implying a common catalytic mechanism. However, Rab9 contains seven regions that are significantly different in conformation from other Rab proteins. Some of those regions coincide with putative effector-binding sites and switch I and switch II regions identified by structure/sequence alignments. The Rab9 structure at near atomic resolution provides an excellent model for structure-based antiviral drug design.

About this Structure

1WMS is a Single protein structure of sequence from Homo sapiens with as ligand. Full crystallographic information is available from OCA.

Reference

High resolution crystal structure of human Rab9 GTPase: a novel antiviral drug target., Chen L, DiGiammarino E, Zhou XE, Wang Y, Toh D, Hodge TW, Meehan EJ, J Biol Chem. 2004 Sep 17;279(38):40204-8. Epub 2004 Jul 19. PMID:15263003

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