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2a78

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(New page: 200px<br /> <applet load="2a78" size="450" color="white" frame="true" align="right" spinBox="true" caption="2a78, resolution 1.810&Aring;" /> '''Crystal structure ...)
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'''Crystal structure of the C3bot-RalA complex reveals a novel type of action of a bacterial exoenzyme'''<br />
'''Crystal structure of the C3bot-RalA complex reveals a novel type of action of a bacterial exoenzyme'''<br />
==Overview==
==Overview==
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C3 exoenzymes from bacterial pathogens ADP-ribosylate and inactivate, low-molecular-mass GTPases of the Rho subfamily. Ral, a Ras subfamily, GTPase, binds the C3 exoenzymes from Clostridium botulinum and C. limosum, with high affinity without being a substrate for ADP ribosylation. In the, complex, the ADP-ribosyltransferase activity of C3 is blocked, while, binding of NAD and NAD-glycohydrolase activity remain. Here we report the, crystal structure of C3 from C. botulinum in a complex with GDP-bound RalA, at 1.8 A resolution. C3 binds RalA with a helix-loop-helix motif that is, adjacent to the active site. A quaternary complex with NAD suggests a mode, for ADP-ribosyltransferase inhibition. Interaction of C3 with RalA occurs, at a unique interface formed by the switch-II region, helix alpha3 and the, P loop of the GTPase. C3-binding stabilizes the GDP-bound conformation of, RalA and blocks nucleotide release. Our data indicate that C. botulinum, exoenzyme C3 is a single-domain toxin with bifunctional properties, targeting Rho GTPases by ADP ribosylation and Ral by a guanine nucleotide, dissociation inhibitor-like effect, which blocks nucleotide exchange.
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C3 exoenzymes from bacterial pathogens ADP-ribosylate and inactivate low-molecular-mass GTPases of the Rho subfamily. Ral, a Ras subfamily GTPase, binds the C3 exoenzymes from Clostridium botulinum and C. limosum with high affinity without being a substrate for ADP ribosylation. In the complex, the ADP-ribosyltransferase activity of C3 is blocked, while binding of NAD and NAD-glycohydrolase activity remain. Here we report the crystal structure of C3 from C. botulinum in a complex with GDP-bound RalA at 1.8 A resolution. C3 binds RalA with a helix-loop-helix motif that is adjacent to the active site. A quaternary complex with NAD suggests a mode for ADP-ribosyltransferase inhibition. Interaction of C3 with RalA occurs at a unique interface formed by the switch-II region, helix alpha3 and the P loop of the GTPase. C3-binding stabilizes the GDP-bound conformation of RalA and blocks nucleotide release. Our data indicate that C. botulinum exoenzyme C3 is a single-domain toxin with bifunctional properties targeting Rho GTPases by ADP ribosylation and Ral by a guanine nucleotide dissociation inhibitor-like effect, which blocks nucleotide exchange.
==About this Structure==
==About this Structure==
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2A78 is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Clostridium_botulinum_d_phage Clostridium botulinum d phage] and [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with MG and GDP as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=2A78 OCA].
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2A78 is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Clostridium_botulinum_d_phage Clostridium botulinum d phage] and [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=MG:'>MG</scene> and <scene name='pdbligand=GDP:'>GDP</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2A78 OCA].
==Reference==
==Reference==
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[[Category: rho]]
[[Category: rho]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 20:46:55 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 16:24:16 2008''

Revision as of 14:24, 21 February 2008


2a78, resolution 1.810Å

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Crystal structure of the C3bot-RalA complex reveals a novel type of action of a bacterial exoenzyme

Overview

C3 exoenzymes from bacterial pathogens ADP-ribosylate and inactivate low-molecular-mass GTPases of the Rho subfamily. Ral, a Ras subfamily GTPase, binds the C3 exoenzymes from Clostridium botulinum and C. limosum with high affinity without being a substrate for ADP ribosylation. In the complex, the ADP-ribosyltransferase activity of C3 is blocked, while binding of NAD and NAD-glycohydrolase activity remain. Here we report the crystal structure of C3 from C. botulinum in a complex with GDP-bound RalA at 1.8 A resolution. C3 binds RalA with a helix-loop-helix motif that is adjacent to the active site. A quaternary complex with NAD suggests a mode for ADP-ribosyltransferase inhibition. Interaction of C3 with RalA occurs at a unique interface formed by the switch-II region, helix alpha3 and the P loop of the GTPase. C3-binding stabilizes the GDP-bound conformation of RalA and blocks nucleotide release. Our data indicate that C. botulinum exoenzyme C3 is a single-domain toxin with bifunctional properties targeting Rho GTPases by ADP ribosylation and Ral by a guanine nucleotide dissociation inhibitor-like effect, which blocks nucleotide exchange.

About this Structure

2A78 is a Protein complex structure of sequences from Clostridium botulinum d phage and Homo sapiens with and as ligands. Full crystallographic information is available from OCA.

Reference

Crystal structure of the C3bot-RalA complex reveals a novel type of action of a bacterial exoenzyme., Pautsch A, Vogelsgesang M, Trankle J, Herrmann C, Aktories K, EMBO J. 2005 Oct 19;24(20):3670-80. Epub 2005 Sep 22. PMID:16177825

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