2aw2

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(New page: 200px<br /> <applet load="2aw2" size="450" color="white" frame="true" align="right" spinBox="true" caption="2aw2, resolution 2.80&Aring;" /> '''Crystal structure o...)
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[[Image:2aw2.gif|left|200px]]<br />
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[[Image:2aw2.gif|left|200px]]<br /><applet load="2aw2" size="350" color="white" frame="true" align="right" spinBox="true"
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<applet load="2aw2" size="450" color="white" frame="true" align="right" spinBox="true"
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caption="2aw2, resolution 2.80&Aring;" />
caption="2aw2, resolution 2.80&Aring;" />
'''Crystal structure of the human BTLA-HVEM complex'''<br />
'''Crystal structure of the human BTLA-HVEM complex'''<br />
==Overview==
==Overview==
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Five CD28-like proteins exert positive or negative effects on immune, cells. Only four of these five receptors interact with members of the B7, family. The exception is BTLA (B and T lymphocyte attenuator), which, instead interacts with the tumor necrosis factor receptor superfamily, member HVEM (herpes virus entry mediator). To better understand this, interaction, we determined the 2.8-A crystal structure of the BTLA-HVEM, complex. This structure shows that BTLA binds the N-terminal cysteine-rich, domain of HVEM and employs a unique binding surface compared with other, CD28-like receptors. Moreover, the structure shows that BTLA recognizes, the same surface on HVEM as gD (herpes virus glycoprotein D) and utilizes, a similar binding motif. Light scattering analysis demonstrates that the, extracellular domain of BTLA is monomeric and that BTLA and HVEM form a, 1:1 complex. Alanine-scanning mutagenesis of HVEM was used to further, define critical binding residues. Finally, BTLA adopts an immunoglobulin, I-set fold. Despite structural similarities to other CD28-like members, BTLA represents a unique co-receptor.
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Five CD28-like proteins exert positive or negative effects on immune cells. Only four of these five receptors interact with members of the B7 family. The exception is BTLA (B and T lymphocyte attenuator), which instead interacts with the tumor necrosis factor receptor superfamily member HVEM (herpes virus entry mediator). To better understand this interaction, we determined the 2.8-A crystal structure of the BTLA-HVEM complex. This structure shows that BTLA binds the N-terminal cysteine-rich domain of HVEM and employs a unique binding surface compared with other CD28-like receptors. Moreover, the structure shows that BTLA recognizes the same surface on HVEM as gD (herpes virus glycoprotein D) and utilizes a similar binding motif. Light scattering analysis demonstrates that the extracellular domain of BTLA is monomeric and that BTLA and HVEM form a 1:1 complex. Alanine-scanning mutagenesis of HVEM was used to further define critical binding residues. Finally, BTLA adopts an immunoglobulin I-set fold. Despite structural similarities to other CD28-like members, BTLA represents a unique co-receptor.
==About this Structure==
==About this Structure==
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2AW2 is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with NI as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=2AW2 OCA].
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2AW2 is a [http://en.wikipedia.org/wiki/Protein_complex Protein complex] structure of sequences from [http://en.wikipedia.org/wiki/Homo_sapiens Homo sapiens] with <scene name='pdbligand=NI:'>NI</scene> as [http://en.wikipedia.org/wiki/ligand ligand]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2AW2 OCA].
==Reference==
==Reference==
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[[Category: Homo sapiens]]
[[Category: Homo sapiens]]
[[Category: Protein complex]]
[[Category: Protein complex]]
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[[Category: Compaan, D.M.]]
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[[Category: Compaan, D M.]]
[[Category: Eaton, D.]]
[[Category: Eaton, D.]]
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[[Category: Gonzalez, L.C.]]
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[[Category: Gonzalez, L C.]]
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[[Category: Hymowitz, S.G.]]
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[[Category: Hymowitz, S G.]]
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[[Category: Loyet, K.M.]]
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[[Category: Loyet, K M.]]
[[Category: Tom, I.]]
[[Category: Tom, I.]]
[[Category: NI]]
[[Category: NI]]
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[[Category: tnfrsf]]
[[Category: tnfrsf]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Mon Nov 12 20:55:15 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 16:31:34 2008''

Revision as of 14:31, 21 February 2008


2aw2, resolution 2.80Å

Drag the structure with the mouse to rotate

Crystal structure of the human BTLA-HVEM complex

Overview

Five CD28-like proteins exert positive or negative effects on immune cells. Only four of these five receptors interact with members of the B7 family. The exception is BTLA (B and T lymphocyte attenuator), which instead interacts with the tumor necrosis factor receptor superfamily member HVEM (herpes virus entry mediator). To better understand this interaction, we determined the 2.8-A crystal structure of the BTLA-HVEM complex. This structure shows that BTLA binds the N-terminal cysteine-rich domain of HVEM and employs a unique binding surface compared with other CD28-like receptors. Moreover, the structure shows that BTLA recognizes the same surface on HVEM as gD (herpes virus glycoprotein D) and utilizes a similar binding motif. Light scattering analysis demonstrates that the extracellular domain of BTLA is monomeric and that BTLA and HVEM form a 1:1 complex. Alanine-scanning mutagenesis of HVEM was used to further define critical binding residues. Finally, BTLA adopts an immunoglobulin I-set fold. Despite structural similarities to other CD28-like members, BTLA represents a unique co-receptor.

About this Structure

2AW2 is a Protein complex structure of sequences from Homo sapiens with as ligand. Full crystallographic information is available from OCA.

Reference

Attenuating lymphocyte activity: the crystal structure of the BTLA-HVEM complex., Compaan DM, Gonzalez LC, Tom I, Loyet KM, Eaton D, Hymowitz SG, J Biol Chem. 2005 Nov 25;280(47):39553-61. Epub 2005 Sep 16. PMID:16169851

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