2fez

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(New page: 200px<br /><applet load="2fez" size="450" color="white" frame="true" align="right" spinBox="true" caption="2fez, resolution 2.00&Aring;" /> '''Mycobacterium tuberc...)
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[[Image:2fez.gif|left|200px]]<br /><applet load="2fez" size="350" color="white" frame="true" align="right" spinBox="true"
caption="2fez, resolution 2.00&Aring;" />
caption="2fez, resolution 2.00&Aring;" />
'''Mycobacterium tuberculosis EmbR'''<br />
'''Mycobacterium tuberculosis EmbR'''<br />
==Overview==
==Overview==
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Ser/Thr phosphorylation has emerged as a critical regulatory mechanism in, a number of bacteria, including Mycobacterium tuberculosis. This, problematic pathogen encodes 11 eukaryotic-like Ser/Thr kinases, yet few, substrates or signaling targets have been characterized. Here, we report, the structure of EmbR (2.0 A), a putative transcriptional regulator of key, arabinosyltransferases (EmbC, -A, and -B), and an endogenous substrate of, the Ser/Thr-kinase PknH. EmbR presents a unique domain architecture: the, N-terminal winged-helix DNA-binding domain forms an extensive interface, with the all-helical central bacterial transcriptional activation domain, and is positioned adjacent to the regulatory C-terminal, forkhead-associated (FHA) domain, which mediates binding to a, Thr-phosphorylated site in PknH. The structure in complex with a, phospho-peptide (1.9 A) reveals a conserved mode of phospho-threonine, recognition by the FHA domain and evidence for specific recognition of the, cognate kinase. The present structures suggest hypotheses as to how EmbR, might propagate the phospho-relay signal from its cognate kinase, while, serving as a template for the structurally uncharacterized Streptomyces, antibiotic regulatory protein family of transcription factors.
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Ser/Thr phosphorylation has emerged as a critical regulatory mechanism in a number of bacteria, including Mycobacterium tuberculosis. This problematic pathogen encodes 11 eukaryotic-like Ser/Thr kinases, yet few substrates or signaling targets have been characterized. Here, we report the structure of EmbR (2.0 A), a putative transcriptional regulator of key arabinosyltransferases (EmbC, -A, and -B), and an endogenous substrate of the Ser/Thr-kinase PknH. EmbR presents a unique domain architecture: the N-terminal winged-helix DNA-binding domain forms an extensive interface with the all-helical central bacterial transcriptional activation domain and is positioned adjacent to the regulatory C-terminal forkhead-associated (FHA) domain, which mediates binding to a Thr-phosphorylated site in PknH. The structure in complex with a phospho-peptide (1.9 A) reveals a conserved mode of phospho-threonine recognition by the FHA domain and evidence for specific recognition of the cognate kinase. The present structures suggest hypotheses as to how EmbR might propagate the phospho-relay signal from its cognate kinase, while serving as a template for the structurally uncharacterized Streptomyces antibiotic regulatory protein family of transcription factors.
==About this Structure==
==About this Structure==
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2FEZ is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Mycobacterium_tuberculosis Mycobacterium tuberculosis]. Full crystallographic information is available from [http://ispc.weizmann.ac.il/oca-bin/ocashort?id=2FEZ OCA].
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2FEZ is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Mycobacterium_tuberculosis Mycobacterium tuberculosis]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2FEZ OCA].
==Reference==
==Reference==
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[[Category: Mycobacterium tuberculosis]]
[[Category: Mycobacterium tuberculosis]]
[[Category: Single protein]]
[[Category: Single protein]]
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[[Category: Alderwick, L.J.]]
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[[Category: Alderwick, L J.]]
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[[Category: Besra, G.S.]]
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[[Category: Besra, G S.]]
[[Category: Futterer, K.]]
[[Category: Futterer, K.]]
[[Category: transcriptional regulator; winged-helix; tetratricopeptide repeat; beta-sandwich;]]
[[Category: transcriptional regulator; winged-helix; tetratricopeptide repeat; beta-sandwich;]]
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''Page seeded by [http://ispc.weizmann.ac.il/oca OCA ] on Wed Nov 21 10:31:27 2007''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 17:20:47 2008''

Revision as of 15:20, 21 February 2008


2fez, resolution 2.00Å

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Mycobacterium tuberculosis EmbR

Overview

Ser/Thr phosphorylation has emerged as a critical regulatory mechanism in a number of bacteria, including Mycobacterium tuberculosis. This problematic pathogen encodes 11 eukaryotic-like Ser/Thr kinases, yet few substrates or signaling targets have been characterized. Here, we report the structure of EmbR (2.0 A), a putative transcriptional regulator of key arabinosyltransferases (EmbC, -A, and -B), and an endogenous substrate of the Ser/Thr-kinase PknH. EmbR presents a unique domain architecture: the N-terminal winged-helix DNA-binding domain forms an extensive interface with the all-helical central bacterial transcriptional activation domain and is positioned adjacent to the regulatory C-terminal forkhead-associated (FHA) domain, which mediates binding to a Thr-phosphorylated site in PknH. The structure in complex with a phospho-peptide (1.9 A) reveals a conserved mode of phospho-threonine recognition by the FHA domain and evidence for specific recognition of the cognate kinase. The present structures suggest hypotheses as to how EmbR might propagate the phospho-relay signal from its cognate kinase, while serving as a template for the structurally uncharacterized Streptomyces antibiotic regulatory protein family of transcription factors.

About this Structure

2FEZ is a Single protein structure of sequence from Mycobacterium tuberculosis. Full crystallographic information is available from OCA.

Reference

Molecular structure of EmbR, a response element of Ser/Thr kinase signaling in Mycobacterium tuberculosis., Alderwick LJ, Molle V, Kremer L, Cozzone AJ, Dafforn TR, Besra GS, Futterer K, Proc Natl Acad Sci U S A. 2006 Feb 21;103(8):2558-63. Epub 2006 Feb 13. PMID:16477027

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