2rmi

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(New page: 200px<br /><applet load="2rmi" size="350" color="white" frame="true" align="right" spinBox="true" caption="2rmi" /> '''3D NMR structure of astressin'''<br /> ==Ov...)
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==Overview==
==Overview==
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The C-terminally amidated CRF antagonist astressin binds to CRF-R1 or, CRF-R2 receptors with low nanomolar affinity while the corresponding, astressin-acid has &gt;100 times less affinity. To understand the role of the, amide group in binding, the conformations of astressin-amide and, astressin-acid were studied in DMSO using NMR techniques. The 3D NMR, structures show that the backbones of both analogs prefer an alpha-helical, conformation, with a small kink around Gln(26). However, astressin-amide, has a well-defined helical structure from Leu(27) to Ile(41) and a, conformation very similar to the bioactive conformation reported by our, group (Grace et al., Proc Natl Acad Sci USA 2007, 104, 4858-4863). In, contrast, astressin-acid has an irregular helical conformation from, Arg(35) onward, including a rearrangement of the side chains in that, region. This structural difference highlights the crucial role of the, C-terminal amidation for stabilization of astressin's bioactive, conformation.
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The C-terminally amidated CRF antagonist astressin binds to CRF-R1 or CRF-R2 receptors with low nanomolar affinity while the corresponding astressin-acid has &gt;100 times less affinity. To understand the role of the amide group in binding, the conformations of astressin-amide and astressin-acid were studied in DMSO using NMR techniques. The 3D NMR structures show that the backbones of both analogs prefer an alpha-helical conformation, with a small kink around Gln(26). However, astressin-amide has a well-defined helical structure from Leu(27) to Ile(41) and a conformation very similar to the bioactive conformation reported by our group (Grace et al., Proc Natl Acad Sci USA 2007, 104, 4858-4863). In contrast, astressin-acid has an irregular helical conformation from Arg(35) onward, including a rearrangement of the side chains in that region. This structural difference highlights the crucial role of the C-terminal amidation for stabilization of astressin's bioactive conformation.
==About this Structure==
==About this Structure==
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[[Category: Riek, R.]]
[[Category: Riek, R.]]
[[Category: Rivier, J.]]
[[Category: Rivier, J.]]
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[[Category: Royappa, G.C.R.]]
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[[Category: Royappa, G C.R.]]
[[Category: astressin]]
[[Category: astressin]]
[[Category: crf antagonist]]
[[Category: crf antagonist]]
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[[Category: urotensins]]
[[Category: urotensins]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Wed Jan 23 12:38:50 2008''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 18:48:29 2008''

Revision as of 16:48, 21 February 2008


2rmi

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3D NMR structure of astressin

Overview

The C-terminally amidated CRF antagonist astressin binds to CRF-R1 or CRF-R2 receptors with low nanomolar affinity while the corresponding astressin-acid has >100 times less affinity. To understand the role of the amide group in binding, the conformations of astressin-amide and astressin-acid were studied in DMSO using NMR techniques. The 3D NMR structures show that the backbones of both analogs prefer an alpha-helical conformation, with a small kink around Gln(26). However, astressin-amide has a well-defined helical structure from Leu(27) to Ile(41) and a conformation very similar to the bioactive conformation reported by our group (Grace et al., Proc Natl Acad Sci USA 2007, 104, 4858-4863). In contrast, astressin-acid has an irregular helical conformation from Arg(35) onward, including a rearrangement of the side chains in that region. This structural difference highlights the crucial role of the C-terminal amidation for stabilization of astressin's bioactive conformation.

About this Structure

2RMI is a Single protein structure of sequence from [1]. Full crystallographic information is available from OCA.

Reference

Astressin-amide and astressin-acid are structurally different in dimethylsulfoxide., Grace CR, Cervini L, Gulyas J, Rivier J, Riek R, Biopolymers. 2007 Oct 5-15;87(2-3):196-205. PMID:17657708

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