2mmz

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m (Protected "2mmz" [edit=sysop:move=sysop])
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'''Unreleased structure'''
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{{STRUCTURE_2mmz| PDB=2mmz | SCENE= }}
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===Solution structure of the apo form of human glutaredoxin 5===
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The entry 2mmz is ON HOLD
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==Disease==
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[[http://www.uniprot.org/uniprot/GLRX5_HUMAN GLRX5_HUMAN]] Adult-onset autosomal recessive sideroblastic anemia. The disease is caused by mutations affecting the gene represented in this entry.
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Authors: Banci, L., Brancaccio, D., Ciofi-Baffoni, S., Del Conte, R., Gadepalli, R., Mikolajczyk, M., Neri, S., Piccioli, M., Winkelmann, J.
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==Function==
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[[http://www.uniprot.org/uniprot/GLRX5_HUMAN GLRX5_HUMAN]] Monothiol glutaredoxin involved in the biogenesis of iron-sulfur clusters. Required for normal iron homeostasis. Required for normal regulation of hemoglobin synthesis by the iron-sulfur protein ACO1.
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Description: Solution structure of the apo form of human glutaredoxin 5
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==About this Structure==
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[[2mmz]] is a 1 chain structure. Full experimental information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2MMZ OCA].
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[[Category: Banci, L.]]
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[[Category: Brancaccio, D.]]
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[[Category: Ciofi-Baffoni, S.]]
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[[Category: Conte, R Del.]]
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[[Category: Gadepalli, R.]]
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[[Category: Mikolajczyk, M.]]
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[[Category: Neri, S.]]
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[[Category: Piccioli, M.]]
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[[Category: Winkelmann, J.]]
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[[Category: Iron-sulfur protein biogenesis]]
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[[Category: Metal binding protein]]
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[[Category: Monothiol glutaredoxin]]

Revision as of 09:54, 16 April 2014

Template:STRUCTURE 2mmz

Contents

Solution structure of the apo form of human glutaredoxin 5

Disease

[GLRX5_HUMAN] Adult-onset autosomal recessive sideroblastic anemia. The disease is caused by mutations affecting the gene represented in this entry.

Function

[GLRX5_HUMAN] Monothiol glutaredoxin involved in the biogenesis of iron-sulfur clusters. Required for normal iron homeostasis. Required for normal regulation of hemoglobin synthesis by the iron-sulfur protein ACO1.

About this Structure

2mmz is a 1 chain structure. Full experimental information is available from OCA.

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OCA

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