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2beo

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[[Image:2beo.gif|left|200px]]<br /><applet load="2beo" size="350" color="white" frame="true" align="right" spinBox="true"
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[[Image:2beo.gif|left|200px]]
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caption="2beo, resolution 2.70&Aring;" />
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'''PRFA, TRANSCRIPTIONAL REGULATOR IN LISTERIA MONOCYTOGENES'''<br />
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{{Structure
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|PDB= 2beo |SIZE=350|CAPTION= <scene name='initialview01'>2beo</scene>, resolution 2.70&Aring;
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|SITE= <scene name='pdbsite=AC1:GLN+Binding+Site+For+Chain+A'>AC1</scene>
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|LIGAND= <scene name='pdbligand=CL:CHLORIDE+ION'>CL</scene>, <scene name='pdbligand=GLN:GLUTAMINE'>GLN</scene>, <scene name='pdbligand=PG4:TETRAETHYLENE+GLYCOL'>PG4</scene> and <scene name='pdbligand=ACM:ACETAMIDE'>ACM</scene>
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|ACTIVITY=
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|GENE=
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}}
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'''PRFA, TRANSCRIPTIONAL REGULATOR IN LISTERIA MONOCYTOGENES'''
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==Overview==
==Overview==
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==About this Structure==
==About this Structure==
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2BEO is a [http://en.wikipedia.org/wiki/Single_protein Single protein] structure of sequence from [http://en.wikipedia.org/wiki/Listeria_monocytogenes Listeria monocytogenes] with <scene name='pdbligand=CL:'>CL</scene>, <scene name='pdbligand=GLN:'>GLN</scene>, <scene name='pdbligand=PG4:'>PG4</scene> and <scene name='pdbligand=ACM:'>ACM</scene> as [http://en.wikipedia.org/wiki/ligands ligands]. Known structural/functional Site: <scene name='pdbsite=AC1:GLN+Binding+Site+For+Chain+A'>AC1</scene>. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2BEO OCA].
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2BEO is a [[Single protein]] structure of sequence from [http://en.wikipedia.org/wiki/Listeria_monocytogenes Listeria monocytogenes]. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=2BEO OCA].
==Reference==
==Reference==
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The mutation G145S in PrfA, a key virulence regulator of Listeria monocytogenes, increases DNA-binding affinity by stabilizing the HTH motif., Eiting M, Hageluken G, Schubert WD, Heinz DW, Mol Microbiol. 2005 Apr;56(2):433-46. PMID:[http://ispc.weizmann.ac.il//pmbin/getpm?pmid=15813735 15813735]
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The mutation G145S in PrfA, a key virulence regulator of Listeria monocytogenes, increases DNA-binding affinity by stabilizing the HTH motif., Eiting M, Hageluken G, Schubert WD, Heinz DW, Mol Microbiol. 2005 Apr;56(2):433-46. PMID:[http://www.ncbi.nlm.nih.gov/pubmed/15813735 15813735]
[[Category: Listeria monocytogenes]]
[[Category: Listeria monocytogenes]]
[[Category: Single protein]]
[[Category: Single protein]]
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[[Category: virulence]]
[[Category: virulence]]
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Feb 21 16:37:00 2008''
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''Page seeded by [http://oca.weizmann.ac.il/oca OCA ] on Thu Mar 20 16:00:56 2008''

Revision as of 14:00, 20 March 2008


PDB ID 2beo

Drag the structure with the mouse to rotate
, resolution 2.70Å
Sites:
Ligands: , , and
Coordinates: save as pdb, mmCIF, xml



PRFA, TRANSCRIPTIONAL REGULATOR IN LISTERIA MONOCYTOGENES


Overview

Listeria monocytogenes, a Gram-positive, facultative intracellular human pathogen, causes systemic infections with high mortality rate. The majority of the known pathogenicity factors of L. monocytogenes is regulated by a single transcription factor, PrfA. Hyperhaemolytic laboratory strains of L. monocytogenes express the constitutively active mutant PrfA(G145S) inducing virulence gene overexpression independent of environmental conditions. PrfA belongs to the Crp/Fnr family of transcription factors generally activated by a small effector, such as cAMP or O(2). We present the crystal structures of wild-type PrfA, the first Gram-positive member of the Crp/Fnr family, and of the constitutively active mutant PrfA(G145S). Cap (Crp) has previously been described exclusively in the cAMP-induced (DNA-free and -bound) conformation. By contrast, the PrfA structures present views both of the non-induced state and of the mutationally activated form. The low DNA-binding affinity of wild-type PrfA is supported both structurally (partly disordered helix-turn-helix motif, overall geometry of the HTH alpha-helices deviates from Cap) and by surface plasmon resonance analyses (K(D) = 0.9 microM). In PrfA(G145S) the HTH motifs dramatically rearrange to adopt a conformation comparable to cAMP-induced Cap and hence favourable for DNA binding, supported by a DNA-binding affinity of 50 nM. Finally, the hypothesis that wild-type PrfA, like other Crp/Fnr family members, may require an as yet unidentified cofactor for activation is supported by the presence of a distinct tunnel in PrfA, located at the interface of the beta-barrel and the DNA-binding domain.

About this Structure

2BEO is a Single protein structure of sequence from Listeria monocytogenes. Full crystallographic information is available from OCA.

Reference

The mutation G145S in PrfA, a key virulence regulator of Listeria monocytogenes, increases DNA-binding affinity by stabilizing the HTH motif., Eiting M, Hageluken G, Schubert WD, Heinz DW, Mol Microbiol. 2005 Apr;56(2):433-46. PMID:15813735

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