5wp6

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'''Unreleased structure'''
 
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The entry 5wp6 is ON HOLD until Paper Publication
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==Cryo-EM structure of a human TRPM4 channel in complex with calcium and decavanadate==
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<StructureSection load='5wp6' size='340' side='right' caption='[[5wp6]], [[Resolution|resolution]] 3.80&Aring;' scene=''>
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Authors:
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== Structural highlights ==
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<table><tr><td colspan='2'>[[5wp6]] is a 4 chain structure. Full crystallographic information is available from [http://oca.weizmann.ac.il/oca-bin/ocashort?id=5WP6 OCA]. For a <b>guided tour on the structure components</b> use [http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=5WP6 FirstGlance]. <br>
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Description:
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</td></tr><tr id='ligand'><td class="sblockLbl"><b>[[Ligand|Ligands:]]</b></td><td class="sblockDat"><scene name='pdbligand=DVT:DECAVANADATE'>DVT</scene></td></tr>
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[[Category: Unreleased Structures]]
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<tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[http://oca.weizmann.ac.il/oca-docs/fgij/fg.htm?mol=5wp6 FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=5wp6 OCA], [http://pdbe.org/5wp6 PDBe], [http://www.rcsb.org/pdb/explore.do?structureId=5wp6 RCSB], [http://www.ebi.ac.uk/pdbsum/5wp6 PDBsum], [http://prosat.h-its.org/prosat/prosatexe?pdbcode=5wp6 ProSAT]</span></td></tr>
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</table>
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== Disease ==
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[[http://www.uniprot.org/uniprot/TRPM4_HUMAN TRPM4_HUMAN]] Familial progressive cardiac conduction defect;Brugada syndrome. The disease is caused by mutations affecting the gene represented in this entry.
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== Function ==
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[[http://www.uniprot.org/uniprot/TRPM4_HUMAN TRPM4_HUMAN]] Calcium-activated non selective (CAN) cation channel that mediates membrane depolarization. While it is activated by increase in intracellular Ca(2+), it is impermeable to it. Mediates transport of monovalent cations (Na(+) > K(+) > Cs(+) > Li(+)), leading to depolarize the membrane. It thereby plays a central role in cadiomyocytes, neurons from entorhinal cortex, dorsal root and vomeronasal neurons, endocrine pancreas cells, kidney epithelial cells, cochlea hair cells etc. Participates in T-cell activation by modulating Ca(2+) oscillations after T lymphocyte activation, which is required for NFAT-dependent IL2 production. Involved in myogenic constriction of cerebral arteries. Controls insulin secretion in pancreatic beta-cells. May also be involved in pacemaking or could cause irregular electrical activity under conditions of Ca(2+) overload. Affects T-helper 1 (Th1) and T-helper 2 (Th2) cell motility and cytokine production through differential regulation of calcium signaling and NFATC1 localization. Enhances cell proliferation through up-regulation of the beta-catenin signaling pathway.<ref>PMID:12015988</ref> <ref>PMID:12799367</ref> <ref>PMID:15121803</ref> <ref>PMID:15472118</ref> <ref>PMID:15550671</ref> <ref>PMID:16806463</ref> <ref>PMID:20625999</ref> <ref>PMID:20656926</ref>
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== References ==
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<references/>
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__TOC__
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</StructureSection>
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[[Category: Du, J]]
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[[Category: Huang, Y]]
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[[Category: Lu, W]]
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[[Category: Sun, W]]
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[[Category: Winkler, P A]]
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[[Category: Ion channel]]
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[[Category: Membrane protein]]

Revision as of 06:59, 13 December 2017

Cryo-EM structure of a human TRPM4 channel in complex with calcium and decavanadate

5wp6, resolution 3.80Å

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