8jbf

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 <tr id='resources'><td class="sblockLbl"><b>Resources:</b></td><td class="sblockDat"><span class='plainlinks'>[https://proteopedia.org/fgij/fg.htm?mol=8jbf FirstGlance], [http://oca.weizmann.ac.il/oca-bin/ocaids?id=8jbf OCA], [https://pdbe.org/8jbf PDBe], [https://www.rcsb.org/pdb/explore.do?structureId=8jbf RCSB], [https://www.ebi.ac.uk/pdbsum/8jbf PDBsum], [https://prosat.h-its.org/prosat/prosatexe?pdbcode=8jbf ProSAT]</span></td></tr>
 </table>
-== Disease ==
-[https://www.uniprot.org/uniprot/NK3R_HUMAN NK3R_HUMAN] Normosmic congenital hypogonadotropic hypogonadism;Kallmann syndrome. The disease is caused by variants affecting distinct genetic loci, including the gene represented in this entry. The genetics of hypogonadotropic hypogonadism involves various modes of transmission. Oligogenic inheritance has been reported in some patients carrying mutations in TACR3 as well as in other HH-associated genes including FGFR1, SPRY4 and KAL1 (PubMed:23643382).<ref>PMID:23643382</ref>
-== Function ==
-[https://www.uniprot.org/uniprot/NK3R_HUMAN NK3R_HUMAN] This is a receptor for the tachykinin neuropeptide neuromedin-K (neurokinin B). It is associated with G proteins that activate a phosphatidylinositol-calcium second messenger system. The rank order of affinity of this receptor to tachykinins is: neuromedin-K > substance K > substance P.
-== References ==
-<references/>
 __TOC__
 </StructureSection>