Structural highlights
Function
[GLR33_ARATH] Glutamate-gated receptor that probably acts as non-selective cation channel, at least in roots and hypocotyls. Can be triggered by Ala, Asn, Cys, Glu, Gly, Ser and glutathione (a tripeptide consisting of Glu-Gly-Cys). Mediates leaf-to-leaf wound signaling. May be involved in light-signal transduction and calcium homeostasis via the regulation of calcium influx into cells.[1] [2] [3]
References
- ↑ Qi Z, Stephens NR, Spalding EP. Calcium entry mediated by GLR3.3, an Arabidopsis glutamate receptor with a broad agonist profile. Plant Physiol. 2006 Nov;142(3):963-71. doi: 10.1104/pp.106.088989. Epub 2006 Sep , 29. PMID:17012403 doi:http://dx.doi.org/10.1104/pp.106.088989
- ↑ Stephens NR, Qi Z, Spalding EP. Glutamate receptor subtypes evidenced by differences in desensitization and dependence on the GLR3.3 and GLR3.4 genes. Plant Physiol. 2008 Feb;146(2):529-38. doi: 10.1104/pp.107.108134. Epub 2007 Dec , 27. PMID:18162597 doi:http://dx.doi.org/10.1104/pp.107.108134
- ↑ Mousavi SA, Chauvin A, Pascaud F, Kellenberger S, Farmer EE. GLUTAMATE RECEPTOR-LIKE genes mediate leaf-to-leaf wound signalling. Nature. 2013 Aug 22;500(7463):422-6. doi: 10.1038/nature12478. PMID:23969459 doi:http://dx.doi.org/10.1038/nature12478