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Lapatinib

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! colspan="6" align="center"| Tyrosine Kinase Inhibitor [[Pharmaceutical_Drugs#Pharmacokinetics_Translated|Pharmacokinetics]] <ref>R. Khosravan, et al. General Poster Session, Developmental Therapeutics: Cytotoxic Chemotherapy, J Clin Oncol 26: 2008 (May 20 suppl; abstr 2578)</ref><ref>PMID:16133532</ref><ref>PMID:20821331</ref><ref>PMID:20717111</ref>
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! colspan="6" align="center"| VEGFR Inhibitor [[Pharmaceutical_Drugs#Pharmacokinetics_Translated|Pharmacokinetics]] <ref>R. Khosravan, et al. General Poster Session, Developmental Therapeutics: Cytotoxic Chemotherapy, J Clin Oncol 26: 2008 (May 20 suppl; abstr 2578)</ref><ref>PMID:16133532</ref><ref>PMID:20821331</ref><ref>PMID:20717111</ref>
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! Parameter

Revision as of 08:34, 6 December 2010

Pharmacokinetics

VEGFR Inhibitor Pharmacokinetics [1][2][3][4]
Parameter Sunitinib (Sutent) Sorafenib (Nexavar)
Tmax (hr) 4 8
Cmax (ng/ml) 115 24.6
Bioavailability (%) Lapatinib (Tykerb) Sunitinib (Sutent)
Protein Binding (%) 99 95
T1/2 (hr) 9.6 83
AUC (ng/ml/hr) 1429 1921
IC50 (nM) Lapatinib (Tykerb) Sunitinib (Sutent)
Dosage (mg) 100 50
Metabolism Hepatic (CYP3A4) Hepatic (CYP3A4)

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